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Wnt3a activates certain genes in hair follicle cells, including a newly discovered one, EP2, which may affect hair growth.

Human hair follicles produce and respond to erythropoietin, helping protect against stress.

Ectodysplasin inhibits Wnt signaling to help form hair follicles.

Both gene and non-gene areas of DNA evolved to make some mammals hairless.

The gene Ascl4 is not necessary for the development of hair, teeth, or mammary glands.

Hairlessness in mammals is due to complex genetic changes in both genes and regulatory regions.

Dermal factors are crucial in regulating melanin production in skin.

Mitochondrial dysfunction is linked to various skin conditions and could be a target for treatments.

Radiation mainly affects keratinocyte stem cells, not melanocyte stem cells, causing hair to gray.

Estrogens significantly influence hair growth by interacting with receptors in hair follicles and may help regulate the hair growth cycle.

The HoxC gene cluster and its enhancers are essential for developing hair and nails in mammals.

ATP-dependent chromatin-remodeling complexes are crucial for gene regulation, cell differentiation, and organ development in mammals.

Hair follicles produce and respond to melatonin, affecting hair growth and sensitivity to estrogen.

The document concludes that understanding the genes and pathways involved in hair growth is crucial for developing treatments for hair diseases.

Minoxidil boosts hair growth by activating PGHS-1.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

Changes in the HR gene have influenced hair growth and may lead to hair loss conditions in humans.

Parathyroid hormone-related protein helps control hair growth phases in mice.

Immune cells affect hair growth and could lead to new hair loss treatments.

Increased PPARGC1α relates to hair thinning in common baldness.

Rabbit skin analysis showed changes in hair growth and identified miRNAs that may regulate hair follicle development.

Dihydrotestosterone treatment on 2D and 3D-cultured skin cells slows down hair growth by affecting certain genes and could be a potential target for hair loss treatment.

Ectodin integrates BMP, SHH, and FGF signals in developing ectodermal organs.

Brain-Derived Neurotrophic Factor (BDNF) slows down hair growth and promotes hair follicle regression.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.

Male genital development is driven by androgen signaling and understanding it could help address congenital anomalies.

Activating β-catenin in certain skin cells speeds up hair growth in mice.

sPLA2-IIA increases growth in hair follicle stem cells and cancer cells, suggesting it could be targeted for hair growth and cancer treatment.

Understanding hair biology is key to developing better treatments for hair and scalp issues.

Blocking the Mitochondrial Pyruvate Carrier causes stress in hair follicles, which can be reduced by an ISR inhibitor.