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TIP39 and PTH2R help control calcium levels and skin cell development.

Different women's hair and skin glands respond to hormones in varied ways, which can cause unwanted hair growth even with normal hormone levels, and more research is needed to treat this effectively.

BMP signaling is essential for the development of touch domes.

Keratin 79 marks a new group of cells that are key for creating and repairing the hair follicle's structure.

miR-22, a type of microRNA, controls hair growth and its overproduction can cause hair loss, while its absence can speed up hair growth.

The document concludes that more research is needed to understand airway repair and to improve tissue engineering for lung treatments.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

Shh and Dhh affect skin development and can cause tumors, while Ihh does not.

Mice without certain skin proteins had abnormal skin and hair development.

Primary cilia affect the size and oil production of eye glands but not the oil's makeup.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

Blocking mTORC1 reduces skin tumor growth in mice.

FoxN1 overexpression in young mice harms immune cell and skin development.

The research suggests new treatments for skin cancer could target specific cell growth pathways.

Parathyroid hormone-related protein helps control hair growth phases in mice.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Hair follicle stem cells can return to their original niche and help regenerate hair.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

K6/ODC transgenic mice are effective for quickly identifying cancer-causing chemicals.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Activating Nrf2 in skin cells causes skin disease similar to chloracne in mice.

A topical BRAF inhibitor, vemurafenib, can speed up wound healing and promote hair growth, especially in diabetic patients.

The conclusion is that hair growth can be improved by activating hair cycles, changing the surrounding environment, healing wounds to create new hair follicles, and using stem cell technology.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

Loss of Fz6 disrupts hair follicle and associated structures' orientation.

MED1 is essential for normal hair growth and maintaining hair follicle stem cells.

Odontogenic keratocysts are caused by abnormal Hedgehog signaling and can lead to tooth and bone issues.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.