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UV radiation may cause DNA changes in skin, certain UVB therapy helps psoriasis, a new gene mutation is linked to mild piebaldism, different immune cells affect psoriasis, a drug promotes hair growth, and some cancer drugs could treat skin barrier issues.

Scientists developed tools to observe hair regeneration in real time and assess skin health, using glowing mice and light-controlled genes.

Brg1 is crucial for keeping hair follicle stem cells and repairing skin, working with the Sonic Hedgehog pathway to promote hair growth.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

MicroRNAs are crucial in controlling cell signaling, affecting cancer and tissue regeneration.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

The conclusion is that using drugs to block the TOR pathway might slow aging and prevent age-related diseases.

The circadian clock affects skin stem cell behavior, impacting aging and cancer risk.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

The timing of when the gene Bmal1 is active affects aging and survival, with its absence during development, not adulthood, leading to premature aging.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

Epigenetic changes control how adult stem cells work and can lead to diseases like cancer if they go wrong.

Too much Smad7 changes skin and hair development by breaking down a protein called β-catenin, leading to more oil glands and fewer hair follicles.

Early detection and intensive treatment of diseases caused by Staphylococcus aureus toxins are crucial for reducing severe health effects.

Damage to skin releases dsRNA, which activates TLR3 and helps in skin and hair follicle regeneration.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

The AUTS2 gene is linked to neurological disorders and may affect human brain development and cognition.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

Scientists created stem cells that can grow hair and skin.

Androgens block hair growth by disrupting cell signals; targeting GSK-3 may help treat hair loss.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

A new mutation in the human glucocorticoid receptor reduces its function and causes resistance to glucocorticoids.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.