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Different types of sun exposure can damage skin cells and affect healing, with chronic exposure being more harmful, and certain immune cells help in the repair process.

The new skin-targeted COVID-19 vaccine creates strong immune responses and could improve vaccination methods.

Tofacitinib improved arthritis and partially improved hair loss in a lupus patient without side effects.

Stress may affect hair growth by influencing hair follicle development and could contribute to hair loss.

A patient with a PLEC mutation has epidermolysis bullosa, muscular dystrophy, and myasthenia gravis, which improved with steroid treatment.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Regulatory T-cells are important for healing and regenerating tissues in various organs by controlling immune responses and aiding stem cells.

Alopecia areata is an autoimmune condition causing hair loss due to the immune system attacking hair follicles, often influenced by genetics and stress.

Cancer treatments targeting specific cells and the immune system can cause skin, mouth, hair, and nail problems, affecting patients' quality of life and treatment adherence.

Alopecia areata involves immune activation in the scalp, suggesting treatments targeting TH1, TH2, and IL-23 pathways.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

Skin grafts on mice can cause an immune response leading to hair loss, useful for studying human hair loss conditions.

Substances from Chinese medicines show promise for immune support and disease prevention, but the way they are processed affects their effectiveness.

Skin's Regulatory T cells are crucial for maintaining skin health and could be targeted to treat immune-related skin diseases and cancer.

Alopecia areata is a genetic and immune-related hair loss condition that is often associated with other autoimmune diseases and does not typically cause permanent damage to hair follicles.

Alopecia areata is caused by immune system attacks on hair follicles, often triggered by viral infections.

Hair follicles help skin immune recovery after UVB exposure.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Skin problems in COVID-19 patients are rare and may be due to the body's complex immune response or blood clotting issues.

Demodex mites can cause skin issues, especially in people with weak immune systems.

Both vitiligo and alopecia areata involve an immune response triggered by stress and specific genes, with treatments targeting this pathway showing potential.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Topical contact sensitizers can treat certain skin conditions by changing the immune response.

Alopecia areata involves immune system imbalances that may lead to depression and anxiety.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

The review suggests that other immune cells besides CD8+ T cells may contribute to alopecia areata and that targeting regulatory cell defects could improve treatment.

Vitamin A affects hair loss and immune response in alopecia areata.

Benign follicular mucinosis involves immune cells attacking hair follicles.