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TAGX-0003 protected hair follicles and reversed alopecia areata in a mouse model.

Targeting specific T cells may help treat alopecia areata.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Innate lymphoid cells type 1 may contribute to alopecia areata.

The study found that a key immune pathway protecting hair follicles is reduced in a mouse model of scarring hair loss.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

Recent discoveries have improved our understanding of hair loss, but challenges in treatment and knowledge among specialists still exist.

People with alopecia areata have fewer IL-10 producing immune cells, which might contribute to the condition.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

Interleukin-15 can help hair growth and protect hair follicles.

BST2 protein and certain T cells increase in early alopecia areata.

A COVID-19 patient with severe hair loss did not improve with hair loss medication after stopping and restarting it due to the infection.

COVID-19 reinfection may trigger alopecia areata.

ILC1-like cells may contribute to hair loss in alopecia areata.

Alopecia areata is a common autoimmune disease affecting hair follicles, with unclear causes and a need for better treatments.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

IL-15 helps maintain hair growth and protects the immune status of hair follicles.

Hair follicles could help develop eye treatments by studying immune responses.

Calcipotriol's effectiveness for treating alopecia areata is unclear and needs more research.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

TYK2 inhibition may help treat alopecia areata by promoting hair growth and reducing immune response.

Alopecia areata involves persistent gene abnormalities and immune activity, even in regrown hair, suggesting a risk of relapse.

Melanocytes may trigger the immune response in alopecia areata, affecting hair regrowth.

Four genes are potential markers for hair loss condition alopecia areata, linked to a specific type of cell death.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

Pediatric alopecia areata is more immune-active than adult cases, suggesting age-specific treatments and potential use of JAK inhibitors.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Linalool in personal care products may contribute to hair loss by damaging hair follicle stem cells and triggering harmful immune responses.

Linalool in personal care products may worsen frontal fibrosing alopecia by damaging hair follicle stem cells and triggering harmful immune responses.