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Frontal fibrosing alopecia causes hair loss at the front hairline, and no effective treatment exists.

Genetic factors alone might not cause pemphigus vulgaris; other factors like birth complications and puberty may trigger it.

Some children who had stem cell transplants developed permanent hair loss, especially when treated with a drug called busulphan.

Skin changes like hair loss, white patches, and nail changes are common in children after bone marrow transplants, often linked to chronic rejection.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

B2m-free HLA variants may be a new class of HLA important in immune responses and diseases.

Despite progress in treatment, the exact cause of Alopecia areata is still unknown.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

The patient with total hair loss did not regrow hair despite treatment, indicating a poor outlook for this type of hair loss.

Alopecia areata is an autoimmune condition causing hair loss due to the immune system attacking hair follicles, often influenced by genetics and stress.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

Lichen Planopilaris is a hair loss condition best treated early with various medications, including hydroxychloroquine, to prevent permanent baldness.

Somatostatin may help protect hair follicles from immune attacks.

Frontal fibrosing alopecia is a hair loss condition in postmenopausal women, similar to lichen planopilaris, with ineffective treatments.

More research is needed to understand the genetic causes of Alopecia areata to develop better treatments.

Lysophospholipids like LPA and S1P are important for hair growth, immune responses, and vascular development, and could be targeted for treating diseases.

Syphilis patients showed abnormal hair root changes, with no difference between primary and secondary stages.

People with alopecia areata often have lower iron levels than healthy people.

Genetics play a role in acne, but how exactly they contribute is not fully understood.

Skin reactions to drugs are common and can be deadly, usually requiring stopping the drug and may be better prevented with genetic testing in the future.

The document concludes that alopecia areata is an autoimmune disease without a definitive cure, but treatments like corticosteroids are commonly used.

Tofacitinib temporarily regrew hair in a man with alopecia, but its effects didn't last.

Alopecia areata involves immune response and gene changes affecting hair loss.

Stem cells can rejuvenate skin, restore hair, and aid in wound healing.

Alopecia Areata is an autoimmune hair loss condition that needs more research for better treatments.

Greying hairs may be protected from alopecia areata.

SA linked to mitochondrial issues and oxidative stress, while AGA involves disrupted hair growth genes.

The skin condition PUPP usually gets better on its own after giving birth and is unlikely to happen again in future pregnancies.