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Certain gene variations in the Vitamin D receptor may increase the risk of chronic hair loss.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Hairless protein can block vitamin D activation in skin cells.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

Vitamin D receptor is crucial for starting hair growth after birth.

Vitamin D is crucial for skin health and managing skin diseases.

A boy with alopecia regrew hair using a vitamin D cream after other treatments failed.

People with alopecia areata often have lower levels of vitamin D, zinc, and folate, but more research is needed to understand if supplements can help treat it.

Environmental factors like diet and vitamin levels, especially Vitamin D, can affect autoimmune diseases differently, with lifestyle changes potentially improving outcomes.

Some vitamins and minerals like vitamin D and iron can help with certain types of hair loss, but more research is needed for others.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

VDR regulation varies by tissue and is crucial for its biological functions.

TGF-β2 plays a key role in human hair growth and development.

Future hair loss treatments should aim to extend hair growth, reactivate resting follicles, reverse shrinkage, and possibly create new follicles, with gene therapy showing promise.

New biopsy techniques and tools improve alopecia diagnosis, and both too much and too little selenium can cause hair loss.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

Systemic diseases can cause hair loss, which is often reversible with treatment.

A girl had rickets due to a gene mutation affecting vitamin D response.

A vitamin D receptor mutation causes rickets and affects immune responses.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

A 3-year-old with vitamin D-resistant rickets and severe hair loss died despite treatment, highlighting the need for improved management of the condition.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

Early diagnosis and treatment of hereditary vitamin D-resistant rickets (HVDRR) are crucial to prevent growth issues and other health problems.

Two sisters with rickets and hair loss had a genetic issue with vitamin D processing, and only improved when given phosphorus supplements.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.