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Implanting hair-follicle stem cells in mice brains helped repair brain bleeding and reduced brain inflammation.

γδ T cells help with hair growth during wound healing in mice.

Progranulin overexpression leads to shorter, thinner hair and increased cell death in mouse hair follicles.

The genes OVOL1 and OVOL2 are important for hair growth and may be involved in a type of skin tumor.

Valentina Greco's research shows that the environment around hair follicle stem cells is more crucial for regeneration than the stem cells themselves.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Hair loss improved after removing pituitary tumor.

Removing MCPIP1 from myeloid cells in mice leads to hair loss and prevents skin tumors but causes pigmented spots.

Hair follicle stem cells can form both hair follicles and skin.

The antibody created from BCC tissues reacts similarly to both BCC and hair follicles, suggesting BCC may come from hair follicle cells.

Mice with human skin protein K8 had more skin problems and cancer.

Hair loss in elderly women is often caused by various factors, including hormonal changes after menopause.

Inactive hair follicle stem cells help prevent skin cancer.

The silk fibroin-based hydrogel shows promise for treating melanoma and healing infected wounds by killing tumor cells and bacteria, and supporting skin recovery.

A 14-year-old boy is the youngest person reported with a rare, benign tumor related to hair follicles, usually found in older people.

A person had two different benign skin tumors, a trichoadenoma and melanocytic naevi, at the same time but in different places on the face.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

Different types of skin cells have unique genetic markers that affect how likely they are to spread cancer.

Excessive putrescine causes hair loss in transgenic mice by disrupting hair follicle development.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

Hair thinning causes stem cell loss through a process involving Piezo1, calcium, and TNF-α.

Folliculotropic mycosis fungoides can affect the central nervous system in advanced stages.

Cutaneous Lymphadenoma is a unique skin tumor with specific protein markers and common gene mutations that may cause continuous cell growth.

Lacking cyclin D3 reduces skin cancer growth without affecting normal skin cell growth.

DNA methylation is essential for skin and hair follicle development, and could be a target for treating skin diseases.

Hedgehog signaling controls hair follicle development and can affect skin cancer growth.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

The document concludes that identifying the specific cells where skin cancers begin is important for creating better prevention, detection, and treatment methods.