Search

everythinglearnresearchcommunity

for

Search:↓

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

Higher free testosterone levels can increase bone density and decrease body fat but may raise the risk of prostate cancer, hair loss, and benign prostate enlargement.

Some scalp sores are linked to different inherited skin conditions.

Furless male rabbits grew slightly faster and heavier than furred ones, but testosterone levels were not the cause.

Potassium channel openers help form elastic fibers in arteries and can treat elastin deficiency and hypertension.

Dietary essential fatty acids improved skin and hair conditions and partially corrected fat composition in diabetic mice.

Mice genetically modified to produce more Del1 protein had faster hair regrowth.

Genetically modified sheep with more β-catenin grew more wool without changing the wool's length or thickness.

Anti-epileptic drugs may cause visual and hair side effects due to enzyme inhibition, especially in genetically predisposed individuals.

Genetically repaired stem cells may treat certain genetic diseases, Th17 cells are key in fighting systemic fungal infections, hair loss in AGA is due to progenitor cell loss, and α-synuclein transfer might contribute to Parkinson's disease progression.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Psoriasis is a common, genetically influenced skin disease worsened by stress and lifestyle, but targeted treatments are promising.

Acne is genetically linked to a higher risk of depression, anxiety, schizophrenia, and bipolar disorder, with the strongest link to bipolar disorder.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

COVID-19 can lead to different skin symptoms and might trigger autoimmune diseases in genetically susceptible people.

A woman had both Graham Little-Piccardi-Lassueur syndrome, causing hair loss, and complete androgen insensitivity syndrome, making her genetically male but physically female. This suggests androgens don't affect the hair loss condition.

Male pattern baldness is an unintended side effect of the body's use of androgens for muscle growth, especially in those genetically prone to it.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

FPHL is a common, age-related, genetic hair loss with unclear causes and limited treatment options.

Japanese vitiligo patients and their families often have other autoimmune diseases.

Hair grays due to oxidative stress and fewer functioning melanocytes.

Understanding hair follicles through various models can help develop new treatments for hair disorders.

Hair thinning causes stem cell loss through a process involving Piezo1, calcium, and TNF-α.

Women with low SHBG levels and a high 3a-diol G to SHBG ratio are likely to experience female pattern baldness, possibly due to a slight excess of androgens affecting sensitive hair bulbs.

Explosions don't stop hair proteins from being used to identify people.

The best animal model for studying male-pattern baldness is the stumptailed macaque, not rats or mice.

Two siblings both had a rare case of alopecia areata at the same time.

The CUBIC protocol allows detailed 3D visualization of proteins in mouse skin biopsies.

Emerging therapies like stem cell and laser treatments show promise for hair regeneration.

Frontal Fibrosing Alopecia is a hair loss condition mainly affecting postmenopausal women, with unclear causes and various clinical patterns.