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The article concludes that hair loss is a common side effect of drugs treating skin cancer by blocking the hedgehog pathway, but treatment should continue, and more selective drugs might prevent this side effect.

PCOS is a long-term condition that needs more research for better understanding and treatment.

Hair growth environment recreated with challenges; stem cells make successful skin organoids.

Cadmium chloride pollution can cause skin disorders, speed up aging, and prevent hair growth.

Valproic acid can cause hair loss when taken orally but may promote hair growth when applied topically.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

Valproic Acid helps regrow hair in mice and activates a hair growth marker in human cells.

ICAM-1 helps regulate hair growth cycles and skin remodeling.

The study found that the hair loss condition in Cesky Fousek dogs is influenced by multiple genes affecting skin and muscle structure, fat metabolism, and immunity.

Different markers are found in stem cells of the scalp's hair follicle bulge and the surrounding skin.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.

Scarring alopecia affects different hair follicle stem cells than nonscarring alopecia, and the infundibular region could be a new treatment target.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

AGEs and their receptors play a significant role in hair loss by causing inflammation and oxidative stress.

Human alpha defensin 5 helps heal wounds, reduce bacteria, and grow hair on burned skin.

Human hair follicle dermal cells can help repair damaged hair follicles.

The research suggests that p63 and TGF-β1 may help determine tumor type and malignancy in hair follicle and sebaceous tumors.

Adding more glutathione to the preservation solution doesn't reduce oxidative stress in hair transplants.

The research identified specific genes that are active in the cells crucial for hair growth.

Hair loss in Androgenetic Alopecia is not caused by damage to follicular stem cells.

Trichogerminoma is a rare, benign skin tumor from hair cells, with a small risk of becoming cancerous.

Taking 100 mg of DHEA daily for 16 weeks did not change metabolic and reproductive health significantly but increased certain ovarian markers with minimal side effects.

Scientists found markers called CD34 and CD200 that help identify stem cells in mouse and human hair follicles.

A specific mutation in the TRPV3 gene causes hair follicle cells to develop improperly, leading to hair loss.

The TAP flap is effective for treating armpit scars from burns, and tissue-engineered templates with hair follicles can help treat scalp burn alopecia.

UVB radiation harms hair growth and health, causing cell death and other changes in human hair follicles.

Frontal fibrosing alopecia is linked to increased immune system activity and reduced stem cells, suggesting early treatment targeting this pathway might prevent hair follicle damage.

Negative pressure therapy increases hair growth in mice.

Researchers found a new way to isolate sweat glands from the scalp for study and culture.

Stem cells preservation in AGA could be a potential therapy, but more cases needed.