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Stress stops hair growth in mice by causing early hair growth phase end and harmful inflammation through a specific nerve-related pathway.

Stress increases nerve fibers and immune cell activity in mouse skin, possibly worsening skin conditions.

Stress increases a factor in mice that leads to hair loss, and blocking this factor may prevent it.

Certain mice without specific receptors or mast cells don't lose hair from stress.

Stress increases certain chemicals in the skin and nerves, which might worsen skin conditions.

Minoxidil helps prevent stress-caused hair loss in mice.

Three specific proteins can turn adult skin cells into hair-growing cells, suggesting a new hair loss treatment.

The new lab-grown skin model is good for testing sunscreen's protection against DNA damage from UV light.

Dosing time affects finasteride's effectiveness and safety in rats.

Certain drugs can cause early hair growth in mice by affecting the nerves.

Cathepsin L is essential for normal hair growth and development.

Hair follicles produce and respond to melatonin, affecting hair growth and sensitivity to estrogen.

Cytokeratin 19 and cytokeratin 15 are key markers for monitoring the quality and self-renewing potential of engineered skin.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Follistatin helps hair growth and cycling, while activin prevents it.

Stress in mice delays hair growth and treatments blocking substance P can partly reverse this effect.

IFN-γ and IL-2 are important for T cell activation in hair loss in mice.

Certain proteins and their receptors are more active during the growth phase of human hair and could be targeted to treat hair disorders.

Blocking interferon-gamma helps prevent and treat hair loss in Alopecia Areata.

Blocking interferon-gamma might help treat various autoimmune diseases.

The proteins transthyretin and megalin are more present in the growth phase of hair, suggesting they might affect hair health and growth.

Hair follicle melanocytes die during hair regression.

PAD3 plays a key role in hair and skin protein structure and may be linked to skin diseases.

Human skin has less GDNF and its receptor with age.

Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

Calreticulin has roles in healing, immune response, and disease beyond its known functions in the endoplasmic reticulum.

Calcium reduces involucrin in rat hair bulbs but doesn't affect filaggrin and Kdap.

Mutations in Plcd1 and Plcd3 together cause severe hair loss in mice.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

Cyclosporine A was found to increase hair growth in mouse whisker follicles.