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The conclusion is that removing both ovaries is the best treatment for excess male hormones in postmenopausal women, with medication as another option, and managing insulin resistance is important for diagnosis and treatment.

Removing both ovaries can treat increased testosterone and related symptoms in postmenopausal women with ovarian hyperthecosis.

A postmenopausal woman's facial redness, acne, and excess hair were caused by too much hormone therapy for insomnia, but improved after stopping the treatment.

Prostate-specific antigen may be a new marker for excess male hormones in women with polycystic ovary syndrome.

Testosterone affects androgen receptors and lipid storage in cells, while DHEA does not convert to testosterone or affect these receptors in the same way.

Blocking the androgen receptor in skin cells reduces their growth response to male hormones, suggesting a possible treatment for skin conditions linked to androgens.

Red deer only have androgen receptors in neck hair cells for mane growth during breeding season.

Effective treatments for excessive body hair in women include drugs that block androgens, sometimes combined with oral contraceptives.

Anti-androgens can help manage some PCOS symptoms better than metformin but are not more effective than placebos or birth control pills for other symptoms.

FPHL causes hair loss in women due to genetics and hormones; minoxidil and anti-androgens are treatments, and early intervention is advised.

Androgen excess disorders in women were effectively treated with spironolactone, estrogen, and dexamethasone.

Androgens contribute to common male hair loss; more research needed for hair growth medication.

High androgen levels in postmenopausal women may suggest an ovarian tumor, and removing it can improve heart and metabolic health.

Obesity is linked to more severe hair loss in adult men and to excess hair growth, but its effects on hair conditions are complex and need more research.

The bulletin provides guidelines for diagnosing and managing PCOS, suggesting weight loss, hormonal contraceptives, and diabetes screening, with clomiphene for infertility and various treatments for excess hair.

Lifestyle changes can improve body composition, excess male hormone levels, and insulin resistance in women with PCOS.

Spironolactone reduced hair thickness and some testosterone levels in women with excessive hair growth.

Women with excess body hair feel psychological distress influenced by societal beauty standards, but distress isn't directly linked to the amount of hair.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

The document concludes that a systematic approach is crucial to identify causes of androgen excess in women beyond the most common cause, Polycystic ovary syndrome (PCOS).

A postmenopausal woman's hair loss and excess hair growth improved after surgery for ovarian hyperthecosis.

In 2017, a new method was created to efficiently produce a topical antiandrogen, a compound used to treat excess sebum and acne, with a high yield and purity.

Excessive androgen in women can cause acne, hair growth, baldness, and PCOS, often treatable with medication.

Postmenopausal ovary stromal cells have a unique makeup and limited steroid production, suggesting androgens come from the adrenal gland.

Birth control pills help treat acne, especially when caused by excess male hormones, and are safe to use with antibiotics.

Most women with excess hair growth have an underlying hormonal issue, often treated with medication and hair removal methods.

Both estrogens and androgens are important for health in both males and females.

Androgens can both stimulate and cause hair loss, and understanding their effects is key to treating hair disorders.

FAGA diagnosis uses blood tests and trichoscopy, with treatments like topical minoxidil, oral anti-androgens, and hormone-modulating drugs.

Androgens like testosterone are important hormones for both men and women, made differently in each sex and affecting the body by regulating genes and quick interactions with cell components.