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Xiangdong black goats have moderate genetic diversity, minimal inbreeding, and important genes for reproduction, immunity, and other traits.

The research identified key proteins that affect wool fiber thickness in Angora rabbits.

FOXN1 mutations cause severe immunodeficiency, hair loss, nail issues, and thymus defects.

Fox genes are important for hair growth and development in cashmere goats.

Caspases are enzymes important for both cell death and various non-lethal cell functions, affecting head development and hair growth, with different caspases playing specific roles.

Foxn1 gene regulation is crucial for thymus development but not for hair growth.

GPRC5D is linked to the formation of hair, nails, and certain tongue areas.

The HPV type 11 region activates hair-specific gene expression in mice.

Understanding hair follicle development can help improve cashmere quality.

Epidermal stem cells have various roles in skin beyond just maintenance, including forming specialized structures and aiding in skin repair and regeneration.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Stromal Vascular Fraction might help with male sexual dysfunction, but more research is needed to confirm its safety and effectiveness.

Gata6 is important for protecting hair growth cells from DNA damage and keeping normal hair growth.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

Certain microRNAs are linked to various skin diseases and could be used to diagnose and treat these conditions.

Cells treated with Wnt-10b can grow hair after being transplanted into mice.

The study found new details about human hair growth and suggests that preventing a specific biological pathway could potentially treat hair graying.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Wounds heal without scarring in early development but later result in scars, and studying Wnt signaling could help control scarring.

Understanding hair follicle biology and stem cell control could lead to new hair loss treatments.

Mouse zigzag hair bends form due to a 3-day cycle of changes in hair progenitors and their environment.

Minoxidil showed a 30% success rate for hair growth in a study, and various skin treatments were effective, but some had limitations or side effects.

Some viruses can cause cancer by changing cell processes and avoiding the immune system; vaccines and targeted treatments help reduce these cancers.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Topical drugs and near-infrared light therapy show potential for treating alopecia.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.