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The hair follicle infundibulum plays a key role in skin health and disease, and understanding it better could lead to new skin disease treatments.

Vitamin D receptor helps control hair growth genes in skin cells.

A new mutation in the hairless gene causes hair loss and skin wrinkling in mice.

Genetic mutations cause various hair diseases, and whole genome sequencing may reveal more about these conditions.

Trichofolliculoma, a rare benign hair follicle tumor, can potentially recur.

Acne was the most common skin problem in kids, with other conditions like warts and eczema also frequent, varying by age and gender.

Hair follicles are complex, dynamic mini-organs that help us understand cell growth, death, migration, and differentiation, as well as tissue regeneration and tumor biology.

Targeting androgen receptors could be a promising way to treat skin disorders with fewer side effects.

Newborn mouse skin cells can grow hair and this process can be recreated in adult cells to potentially help with hair loss.

Mutant mice help researchers understand hair growth and related genetic factors.

Vitamin D receptor is crucial for starting hair growth after birth.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Hair follicle regeneration possible, more research needed.

Ultrasound is a useful, non-invasive tool in dermatology for diagnosing skin conditions and guiding treatments, but it has some limitations.

The document concludes that over 500 genes are linked to hair disorders and this knowledge is important for creating new treatments.

The proteins transthyretin and megalin are more present in the growth phase of hair, suggesting they might affect hair health and growth.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Lowering testosterone speeds up wound healing in male mice.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

Asian hair loss differs from Europeans; consider individual needs and psychological well-being for treatment.

Pitx2 helps outer root sheath cells differentiate but can't start hair growth on its own.

Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Some hair loss disorders are caused by genetic mutations affecting hair growth.

The mineralocorticoid receptor may play a role in skin and hair health and could be a new target for treating related disorders.