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RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Testosterone can both promote hair growth and cause baldness by affecting hair growth signals.

Rhodococcus equi causes severe pneumonia in young foals, and effective vaccines are needed due to foals' weak immune responses.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

A deficiency in the TTC7A gene causes immune problems, gut issues, and hair loss.

Graft-versus-host disease is a complication where donor immune cells attack the recipient's body, often affecting the skin, liver, and gastrointestinal tract.

Hair aging is caused by stress, hormones, inflammation, and DNA damage affecting hair growth and color.

Nanoparticles can deliver vaccines through hair follicles, triggering immune responses and providing protection.

New therapies are being developed that target integrin pathways to treat various diseases.

Mast cells and CD8 T cells interact closely in skin diseases, affecting each other's behavior and contributing to conditions like psoriasis and eczema.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Double-stranded RNA causes inflammation in hair follicle cells, which may help understand and treat alopecia areata.

The document concludes that pregnancy and cancer share immune evasion tactics, but more research is needed before using checkpoint blockade immunotherapy in pregnant cancer patients to avoid harm to the placenta.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

The MAGE3 hypothesis for alopecia areata did not lead to a significant breakthrough.

R-spondin2 may help treat hair loss, gene differences could explain baldness, a peptide's regulation is linked to psoriasis, B-defensin gene copies may affect a skin condition's risk and severity, and potential markers and targets for alopecia areata were identified.

Botulinum toxin may help reduce stress-related hair loss.

Higher granulysin levels in the blood are linked to more severe hair loss in alopecia areata patients, and these levels decrease after effective treatment.

Increasing Treg cells in the skin does not cure hair loss from alopecia areata in mice.

New treatments for hair growth and psoriasis may be possible, and gene differences could affect baldness and the severity of skin conditions.

Calreticulin has roles in healing, immune response, and disease beyond its known functions in the endoplasmic reticulum.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Fat cells help recruit healing cells and build skin structure during wound healing.

Tofacitinib is safe and effective for severe alopecia areata, but hair loss may return 2 months after stopping treatment.

Ruxolitinib effectively regrows hair in most patients with severe hair loss.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

A patient with Alopecia Areata had complete hair regrowth after using the drug baricitinib.