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Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

Three genes in Arabidopsis are important for plant growth and development by affecting sugar attachment to proteins.

MED1 is essential for normal hair growth and maintaining hair follicle stem cells.

Keratin protein production in cells is controlled by a complex system that changes with cell type, health, and conditions like injury or cancer.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

Understanding hair follicle development can help treat hair loss, skin regeneration, and certain skin cancers.

Skin stem cells interacting with their environment is crucial for maintaining and regenerating skin and hair, and understanding this can help develop new treatments for skin and hair disorders.

TGF-β2 helps activate hair follicle stem cells by counteracting BMP signals.

The Wnt/β-catenin pathway is important for body functions and diseases, and targeting it may treat conditions like cancer, but with safety challenges.

Hair follicles help attract immune cells to the skin during stress.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Epidermolysis bullosa simplex causes easily blistered skin due to faulty skin cell proteins, leading to new treatment ideas.

Cannabinoids may help treat various skin conditions.

Metabolic signals and cell shape influence how cells develop and change.

Androgen receptors are active in many tissues of both male and female mice, not just reproductive organs.

The Hedgehog signaling pathway is important for skin and hair growth and can lead to cancer if it doesn't work right.

Runx1 affects hair growth, cancer development, and autoimmune diseases in epithelial tissues.

Vitamin D and its receptor may help prevent skin cancer.

The project created a standardized system for classifying skin lesions in lab rats and mice.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Macrophage iron release is crucial for hair growth and wound healing.

Neuregulin3 affects cell development in the skin and mammary glands.

Melanoma's complexity requires personalized treatments due to key genetic mutations and tumor-initiating cells.

Odontogenic keratocysts are caused by abnormal Hedgehog signaling and can lead to tooth and bone issues.

The conclusion is that understanding how hair follicle stem cells live or die is important for maintaining healthy tissue and repairing injuries, and could help treat hair loss, but there are still challenges to overcome.

Reducing Tyrosinase prevents mature color pigment cells from forming in mouse hair.

Clim proteins are essential for maintaining healthy corneas and hair follicles.

New hair follicles could be created to treat hair loss.

Improved understanding of stem cell mechanisms can enhance skin tissue engineering.