Search

everythinglearnresearchcommunity

for

Search:↓

Pygo2 is important for early growth and progression of intestinal tumors, and could be a target for treating cancers with certain mutations.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

Hair follicle stem cells are crucial for touch sensation and proper nerve structure in mice.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

The right amount of retinoic acid is essential for normal hair growth and development.

Mutant mice help researchers understand hair growth and related genetic factors.

The document concludes that mouse models are crucial for studying hair biology and that all mutant mice may have hair growth abnormalities that require detailed analysis to identify.

New treatments for diabetes, central nervous system repair, and cartilage injury were found, and a way to create functional hair follicles from stem cells was developed.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.

Cyclooxygenase-2 overexpression in mice skin causes hair loss like human androgenetic alopecia.

Understanding where cancer cells come from helps create better prevention and treatment methods.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Mice can correct hair follicle orientation without certain genes, but proper overall alignment needs those genes.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

Vitamin D receptor helps control hair growth genes in skin cells.

The document concludes that various findings in rheumatology offer insights into disease severity, treatment responses, and potential risks in medication, with some limitations due to unspecified participant numbers.

Blocking Prostaglandin D₂ may help treat hair loss.

D-OCT shows increased blood vessel growth in response to tissue damage in Frontal Fibrosing Alopecia and is useful for diagnosis and monitoring.

Hair growth-related cells need the enzyme SCD1 to help maintain the area that supports hair growth.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

Blocking Prostaglandin D₂ (PGD₂) could help treat hair loss.

Gypenosides from Gynostemma pentaphyllum were found to have anti-aging effects, increasing skin collagen and reducing wrinkles.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

Sirolimus and propranolol may reduce abnormal cell growth and improve lymphatic malformations in children.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.