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The document provides a method to classify human hair growth stages using a model with human scalp on mice, aiming to standardize hair research.

Regenerated fully functional hair follicles using stem cells, with potential for hair regrowth therapy.

The document concludes that the exact way alcohol causes harm to fetal development is unknown, but it significantly affects nutrient transport to the fetus and a safe level of alcohol during pregnancy is not determined.

Hair follicle stem cells are key for hair and skin regeneration, can be reprogrammed, and have potential therapeutic uses, but also carry a risk of cancer.

Chitosan nanofiber scaffolds improve skin healing and are promising for wound treatment.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

Androgens can cause hair growth in some areas and hair loss on the scalp.

Protoporphyrin IX is useful in cancer treatment but can cause health problems if not properly regulated.

Human hair follicle organ culture is a useful model for hair research with potential for studying hair biology and testing treatments.

Mouse hair follicle cells briefly grow during the early hair growth phase, showing that these cells are important for starting the hair cycle.

Prolactin contributes to hair loss by promoting hair follicle shrinkage and cell death.

The telogen phase of hair growth is active and important for preparing hair follicles for regeneration, not just a resting stage.

Applying a special compound can promote hair growth without harmful side effects.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

Too much Smad7 can cause serious changes in skin tissues, including problems with hair growth, thymus shrinkage, and eye development issues.

The document concludes that assessing hair follicle damage due to cyclophosphamide in mice involves analyzing structural changes and suggests a scoring system for standardized evaluation.

Formyl-peptide receptor agonists could be new anti-inflammatory drugs.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

Versican, a protein, is less present in thinning hair follicles and this decrease might contribute to common hair loss in men.

TNF family proteins are crucial for the development of skin features like hair, teeth, and mammary glands.

Hair follicle regeneration needs special conditions and young cells.

Procyanidin compounds from grape seeds were found to significantly increase mouse hair growth.

Zinc deficiency in children can cause skin issues and can be serious if not diagnosed and treated properly.

Men who think they are losing hair feel worse about themselves, especially if they are younger.

S100A4 and S100A6 proteins may activate stem cells for hair follicle regeneration and could be potential targets for hair loss treatments.

Human hair follicles can regenerate after removal, but with low success rate.

Topical drugs and near-infrared light therapy show potential for treating alopecia.

Human hair follicle cells can grow hair when put into mouse skin if they stay in contact with mouse cells.