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The conclusion is that the dermal papilla is crucial for hair follicle regrowth, and hair follicles undergo significant structural changes in addition to cell division during regeneration.

A small group of slow-growing cells causes basal cell carcinoma to return after treatment.

Hair can regrow after certain stem cells are lost because other stem cells can take over their role.

Researchers used a laser to create advanced skin models with hair-like structures.

The study showed that some hair follicle stem cells wake up to grow hair while others stay asleep, and that the environment around them is important for hair growth.

Hair regeneration needs dynamic cell behavior and mesenchyme presence for stem cell activation.

Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.

The hair follicle dermal sheath is essential for hair shedding and needs to communicate with the outer root sheath for normal hair growth cycles.

Mast cells likely promote skin scarring and fibrosis, but their exact role is still unclear.

Certain mature cells in mouse lungs can turn back into stem cells to aid in tissue repair.

Lanceolate complexes in mouse hair follicles are essential for touch and depend on specific cells for maintenance and regeneration.

Hair follicle bulge cells are important for hair survival and help heal the skin after injury, which might be relevant for understanding hidradenitis suppurativa.

Hair follicle dermal stem cells are key for regenerating parts of the hair follicle and determining hair type.

Removing Lrig1-positive stem cells in mice causes temporary loss of sebaceous glands.

More dermal papilla cells in hair follicles lead to larger, healthier hair, while fewer cells cause hair thinning and loss.

The niche environment controls stem cell behavior and plasticity, which is important for tissue health and repair.

The conclusion is that tissue structure is key for stem cell communication and maintaining healthy tissues.

Muscles and nerves that cause goosebumps also help control hair growth.

Skin's epithelial stem cells are crucial for repair and maintenance, and understanding them could improve treatments for skin problems.

Blocking JAK-STAT5 signaling in mice leads to hair growth.

Different types of stem cells with unique roles exist in blood, skin, and intestines, and this variety is important for tissue repair.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

The conclusion is that hair growth can be improved by activating hair cycles, changing the surrounding environment, healing wounds to create new hair follicles, and using stem cell technology.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

Hair thinning causes stem cell loss through a process involving Piezo1, calcium, and TNF-α.

Hair follicle stem cells can return to their original niche and help regenerate hair.

Merkel cells stabilize nerve endings in the skin, and they change independently of each other.

Sirolimus and propranolol may reduce abnormal cell growth and improve lymphatic malformations in children.

Two-photon microscopy helps observe hair follicle stem cell behaviors in mice.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.