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Removing the ATR gene in adult mice causes rapid aging and stem cell loss.

A specific ATR gene mutation is linked to a hereditary oropharyngeal cancer syndrome.

Removing a key gene in mice leads to premature aging and loss of stem cells.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Removing Sprouty genes in mice causes various hormone-related issues but does not increase cancer risk by one year of age.

Maintaining DNA health in stem cells is key to preventing aging and tissue breakdown.

Skin aging is due to impaired stem cell mobilization or fewer responsive stem cells.

Skin aging reflects overall body aging and can indicate internal health conditions.

Deleting the Trf1 protein in mice is safe and may help prevent cancer without major side effects.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

Understanding how melanocyte stem cells work could lead to new treatments for hair graying and skin pigmentation disorders.

The skin protects the body and is constantly renewed by stem cells; disruptions can lead to cancer.

Low oxygen areas help maintain and protect blood stem cells by using a simple sugar breakdown process for energy and managing their activity levels.

Researchers found that certain RNA circles in the brain are linked to disease risk, but their exact role in disease is still unknown.

TRH stimulates human hair growth and extends the hair growth phase.

Mutations in the androgen receptor gene cause different levels of androgen insensitivity, making it hard to create simple tests for the condition.

People with PCNT mutations often develop severe insulin resistance and early-onset diabetes during childhood or adolescence.

PCOS may start before birth, involves metabolic issues, and can be treated with drugs like metformin and lifestyle changes.

The research provides a gene-based framework for hair biology, highlighting the Hippo pathway's importance and suggesting links between hair disorders, cancer pathways, and the immune system.

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

Cancer cells often have more copies of TERT and TERC genes, which helps them grow and could affect patient outcomes.

Cosmetic residues on individual hairs can be identified and differentiated using ATR FT-IR microspectroscopy.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Custom skincare can be made based on genes, fewer cats in Lublin have FeLV/FIV than national average, and studies also looked at small water bodies, river pollution, guppy growth, toxins in biochars, palm oil issues, and pumpkin seed oil for hair strength.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Deleting the Far2 gene in mice causes sebaceous gland issues and patchy hair loss.

Body hairs can be used in forensic science and toxicology like scalp hair.