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ULBP3 levels are higher in Tinea capitis patients and may help predict the disease's severity.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

The technique accurately identifies and evaluates hair follicle structures in skin.

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A woman's sudden hair loss was linked to azathioprine use despite normal enzyme levels, and improved after stopping the drug and starting treatment.

FAGA diagnosis uses blood tests and trichoscopy, with treatments like topical minoxidil, oral anti-androgens, and hormone-modulating drugs.

Dermoscopy effectively diagnoses and evaluates female hair loss treatment.

PRP treatment may promote hair growth and improve hair density in women with AGA, but more research is needed.

Alopecia areata is an autoimmune condition causing hair loss due to the immune system attacking hair follicles, often influenced by genetics and stress.

Alopecia areata is an autoimmune disease causing patchy hair loss, often with other autoimmune disorders, but its exact causes are unknown.

The Lexington LaserComb helped regrow hair in mice with a condition similar to human hair loss.

Trichoscopy is good for telling apart tinea capitis and alopecia areata in kids.

Certain scalp patterns can indicate the severity and activity of hair loss in Turkish alopecia patients.

Certain immune cells contribute to severe hair loss in chronic alopecia areata, with Th17 cells possibly having a bigger impact than cytotoxic T cells.

Less than a quarter of alopecia areata cases were unusual forms or had paradoxical regrowth.

Restoring immune privilege in hair follicles could help treat certain types of hair loss.

Scalp biopsies help tell apart androgenetic alopecia and alopecia areata.

People with alopecia areata have higher levels of osteopontin, which might be important in the disease's development, but this doesn't relate to how severe the disease is.

Alopecia areata treatment should be personalized, using topical or systemic therapies based on severity, with promising options like JAK inhibitors needing more research.

Alopecia areata causes sudden hair loss, has genetic links, and can be managed but not cured.

Stress-related hair loss was reversed with a special medication.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

Hair follicles are normally protected from the immune system, but when this protection fails, it can cause hair loss in alopecia areata.

The most common hair loss type at specialist clinics is androgenetic alopecia, especially in younger men, followed by alopecia areata and telogen effluvium, with differences seen across regions.

Diagnosing diffuse alopecia, a hair loss condition, can be challenging and may require a scalp biopsy or tracking the disease's progression when symptoms and skin tests aren't enough.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

Yellow dots and short vellus hairs are the most common signs of Alopecia Areata (AA), and trichoscopy can help diagnose AA and track treatment progress.

Alopecia areata is an autoimmune disease with genetic links, treatable with certain medications, and can affect mental health.