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Sebaceous glands in male pattern hair loss patients have more lobules and might cause early hair growth phase shifts.

Not all hair loss patients are suitable for hair transplant due to various conditions like unstable hair loss, insufficient hair loss, very young age, unrealistic expectations, certain psychological disorders, and medical unfitness.

The mouse model suggests male pattern baldness may be due to an enzyme increasing DHT and higher androgen receptor levels in hair follicles.

Both minoxidil-spironolactone and minoxidil-finasteride treatments work for hair loss, but minoxidil-spironolactone is slightly more effective, especially in women.

Topical finasteride is promising for hair loss with minimal side effects but needs more research and approval.

Fibrosing alopecia in a pattern distribution is a hair loss condition often confused with other types, requiring early treatment but usually not resulting in significant hair regrowth.

Topical minoxidil is the most promising treatment for male pattern hair loss, but more research is needed to confirm its effectiveness and safety.

Reducing micro-inflammation didn't change hair growth patterns in AGA.

Differences in gene expression and methylation patterns found in AGA patients suggest potential targets for future treatments.

The document explains how hair loss patterns in men and women, known as Androgenetic alopecia, are classified using the Hamilton-Norwood system for men and the Ludwig grade system for women.

Finasteride effectively treats male pattern baldness, improving hair growth and density.

Two gene areas linked to male pattern baldness found, more research needed.

Platelet-rich plasma injections significantly improve hair density and thickness in both male and female pattern hair loss, especially in early stages.

Male pattern baldness may be caused by scalp pressure on hair follicles, which increases with age and leads to a cycle of hair loss. This process is not directly determined by genes.

Male pattern baldness is an unintended side effect of the body's use of androgens for muscle growth, especially in those genetically prone to it.

Female pattern baldness may indicate a higher risk of coronary artery disease.

Older, overweight women with children and a family history of hair loss are more likely to experience female pattern hair loss.

Male pattern baldness, especially the vertex type, could be an early sign of cardiovascular risk factors like high cholesterol and diabetes.

Hair pattern in androgenetic alopecia overlaps with scalp and bone demarcations, with distinct gene profiles affecting susceptibility.

Chronic exposure to sunlight may worsen male pattern baldness and protecting the scalp from the sun could slow it down.

Men with male pattern baldness are more likely to have unhealthy blood lipid levels, which could signal other health issues.

There's no clear link between female pattern hair loss and thickness of the carotid artery, but those with hair loss may have thicker arteries. More research is needed.

Immune activities and specific genes are important in male pattern baldness.

People with hair loss have more androgen receptors and enzymes in certain follicles, with men and women showing different patterns.

Galenic lotions can effectively treat different patterns of hair thinning in men and women.

AR polyglycine repeat doesn't cause baldness.

58% of men aged 30-50 have hair loss, with severity increasing with age.

Finasteride effectively treats hair loss in Korean men for at least 5 years, with V type responding better.

Minoxidil may help treat hair loss by reducing the activity of a specific enzyme linked to baldness.

Baldness, especially at the front, may lower the risk of testicular cancer by 31%, but its link to prostate cancer is unclear.