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Muscle around hair follicles controls hair loss by releasing a signal that causes cell death.

Current hair regeneration methods show promise but face challenges in maintaining cell effectiveness and creating the right environment for hair growth.

Blocking cell death in hair follicles can lead to impaired hair growth.

Small molecule DMF improves psoriasis and multiple sclerosis, adult skin cells can be made to grow new hair, certain skin cells initiate hair growth, IL-17C controls gut health and can cause skin inflammation, and skin cells produce IL-17 that can lead to psoriasis.

Autophagy helps activate hair stem cells and hair growth by changing their energy use to glycolysis.

CD98hc's role in skin health decreases with age.

Scalp psoriasis features reversible hair loss and specific immune activation, with no significant hair follicle damage.

Melanogenesis inhibitors like kojic acid and niacinamide can reduce inflammation and pigment production in skin cells.

Targeting the Smoothened receptor shows promise for treating certain cancers.

Activating Wnt in skin cells controls the number of hair follicles by directing cell movement and fate.

The protein SLC1A3 is important for activating skin stem cells and is necessary for normal hair and skin growth in mice.

SM04755 may be an effective topical treatment for psoriasis.

Behçet's Disease may be caused by genetic and environmental factors leading to abnormal immune responses, and stress management and new treatments could improve patient outcomes.

Aging reduces stem cell activation, leading to hair loss in mice lacking a specific enzyme.

LED light helps human hair root cells grow and prevents them from dying by activating specific growth pathways.

Men are more likely to have severe respiratory viral infections like COVID-19 due to hormonal and genetic differences, while women generally have stronger immune responses.

Platelet Rich Plasma (PRP) shows promise for tissue repair and immune response, but more research is needed to fully understand it and optimize its use.

Blocking β-catenin in skin cells improves hair growth during wound healing.

TCDD disrupts skin stem cells, causing skin issues like chloracne.

Runx1 controls fat-related genes important for normal and cancer cell growth, affecting skin and hair cell behavior.

Activating autophagy in hair follicle stem cells can lead to hair growth and repair by affecting sugar metabolism.

Blocking LFA-1 prevents hair loss in mice.

Integrin alphavbeta6 is important for wound healing and hair growth, and blocking it may improve these processes.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Old people have less hair because their hair follicles don't regenerate as well, not because of fewer stem cells, and a protein called follistatin might help reactivate hair growth.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

Azelaic acid helps protect hair cells from UV damage and encourages hair growth by increasing certain gene expressions and proteins.