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Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

People with X-linked hypohidrotic ectodermal dysplasia have no sweat ducts and less, thinner hair.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

Dlx3 is essential for hair growth and regeneration.

Sostdc1 controls the size and number of hair and mammary gland structures.

The study concluded that PKP1 is essential for skin integrity and hair growth, and its dysfunction causes the symptoms of ectodermal dysplasia/skin fragility syndrome.

Tet2 and Tet3 enzymes are important for controlling hair growth and shape by affecting gene activity and DNA structure in hair follicles.

Estradiol-rich plasma can increase hair density faster and more effectively in male pattern baldness treatment than regular plasma.

Hair mesotherapy might help with certain types of hair loss but lacks strong scientific proof and has some risks.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

Removing HIF-P4H-2 from certain skin cells in mice causes hair loss on the body but not the head.

A specific immune response helps control mite populations on the skin, maintaining healthy hair follicles.

Trichoscopy helps diagnose hairline recession causes in Egyptian women, with androgenetic alopecia being the most common.

A gene mutation in mice causes skin defects and early death.

Proteins like aPKC and PDGF-AA, substances like adenosine and ATP, and adipose-derived stem cells all play important roles in hair growth and health, and could potentially be used to treat hair loss and skin conditions.

Men with baldness due to androgenetic alopecia still have hair stem cells, but lack specific cells needed for hair growth.

The conclusion is that certain scalp tissue changes are characteristic of lichen planopilaris, with mucinous perifollicular fibroplasia being a new feature for diagnosis.

Hair loss in Cronkhite-Canada syndrome is reversible by treating the gut issues and doesn't need steroid treatment for the hair itself.

Certain mice without specific receptors or mast cells don't lose hair from stress.

The gene Ascl4 is not necessary for the development of hair, teeth, or mammary glands.

The study concluded that hair loss treatments should be tailored to the specific type of alopecia and individual patient needs.

Heparan sulfate is important for hair growth, preventing new hair formation in mature skin, and controlling oil gland development.

The protein CCN2 controls hair growth by affecting hair follicle formation and stem cell activity in mice.

Wnt ligands, produced by dermal papilla cells, are essential for adult hair growth and regeneration.

Hair follicle stem cells can help repair nerve and spinal cord injuries.

Human hair follicle organ culture is a useful model for hair research with potential for studying hair biology and testing treatments.

Mouse skin glands need healthy nerves to grow properly during hair growth phases.

Loss of oil glands and inflammation might contribute to the development of scarring hair loss.

Msx2 and Foxn1 are both crucial for hair growth and health.

Regulating keratinocyte growth in engineered skin can improve wound healing.