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Glossary Neurogenic Inflammation

inflammation triggered by nerve-released chemicals causing redness and pain

Neurogenic inflammation is a type of inflammation triggered by the nervous system. It occurs when nerve fibers release certain chemicals, like substance P and calcitonin gene-related peptide (CGRP), which cause blood vessels to expand and immune cells to become activated, leading to redness, swelling, and pain. This process is often involved in conditions like migraines, asthma, and some skin disorders.

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Neogenic Hair Follicles

Research

5 / 1000+ results

research Neurogenic Inflammation in Stress-Induced Termination of Murine Hair Growth Is Promoted by Nerve Growth Factor

108 citations , July 2004 in “American Journal of Pathology”

This study suggests that nerve growth factor plays a crucial role in the stress-induced termination of hair growth in mice, and its antagonism might offer a therapeutic approach to mitigate stress-related hair loss.

research Role of neurogenic inflammation in the pathogenesis of alopecia areata

8 citations , April 2024 in “The Journal of Dermatology”

This study suggests that neuropeptides may contribute to the progression of alopecia areata by causing neurogenic inflammation and disrupting the inflammatory environment of hair follicles, but the exact pathogenesis still requires further investigation.

research Neurogenic skin inflammation in stress‐triggered inhibition of hair growth in mice is promoted via nerve growth factor‐dependent pathways

September 2004 in “Experimental Dermatology”

In this study, neutralizing nerve growth factor reduced stress-triggered hair loss in mice, suggesting that nerve-immune system interactions and neurogenic inflammation contribute to this condition.

research Frontal Fibrosing Alopecia and Increased Scalp Sweating: Is Neurogenic Inflammation the Common Link

15 citations , January 2015 in “Skin Appendage Disorders”

This report discusses a series of patients with frontal fibrosing alopecia experiencing increased scalp sweating, which the authors hypothesize may be related to local skin inflammation affecting sweat gland activity.

research LB1059 Neuromediators associated with neurogenic skin inflammation promote M2 differentiation of macrophages in human skin ex vivo

July 2024 in “Journal of Investigative Dermatology”

Certain substances can help skin cells become anti-inflammatory, aiding in tissue repair.

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community 58M: Nizoral Stopped Working for Scalp Itching, Alternatives?

in General 100 upvotes 11 months ago

A 58-year-old uses finasteride for hair loss and has tried various shampoos, finding Equate T-gel effective for controlling itching. Nizoral was previously effective but no longer works, and DermaKB hasn't been used long enough to assess its effectiveness.

community Oxytocin, empathy, hair, and cinnamon

in General 138 upvotes 2 years ago

Low oxytocin levels in hair are linked to reduced empathy and may affect hair growth. Cinnamic acid can promote hair growth by activating oxytocin receptors.

community Physio-metabolic method of treating androgenic alopecia. Cold receptors. The relationship between DHT, cold receptors, minoxidil and antiandrogens.

2 upvotes 1 year ago

A method for treating androgenic alopecia using minoxidil, antiandrogens, exercise, and cold exposure to promote hair growth. Environmental factors and lifestyle changes, like diet and exercise, can improve treatment effectiveness.

Physio-metabolic method of treating androgenic alopecia. Cold receptors. The relationship between DHT, cold receptors, minoxidil and antiandrogens

community Physio-metabolic method of treating androgenic alopecia. Cold receptors. The relationship between DHT, cold receptors, minoxidil and antiandrogens

in Research 413 upvotes 1 year ago

Treating androgenic alopecia with minoxidil, finasteride, and antiandrogens, alongside exercise, cryotherapy, and natural substances to stimulate cold receptors for better hair growth. The method focuses on enhancing treatment effectiveness by considering environmental and behavioral factors and the role of cold receptors and muscle stress.

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