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Research 121–150 of 495
- Vitamin D Receptor: Key Roles in Bone Mineral Pathophysiology, Molecular Mechanism of Action, and Novel Nutritional Ligands
- Metabolic and cellular analysis of alopecia in vitamin D receptor knockout mice
- Normalization of Mineral Ion Homeostasis by Dietary Means Prevents Hyperparathyroidism, Rickets, and Osteomalacia, But Not Alopecia in Vitamin D Receptor-Ablated Mice
- Targeting Expression of the Human Vitamin D Receptor to the Keratinocytes of Vitamin D Receptor Null Mice Prevents Alopecia
- Evaluation of Keratinocyte Proliferation and Differentiation in Vitamin D Receptor Knockout Mice*
- Mechanism of Vitamin D Receptor Action
- The role of vitamin D receptor mutations in the development of alopecia
- Cloning of a Functional Vitamin D Receptor from the Lamprey (Petromyzon marinus), an Ancient Vertebrate Lacking a Calcified Skeleton and Teeth
- Overexpression of Hedgehog Signaling Is Associated with Epidermal Tumor Formation in Vitamin D Receptor–Null Mice
- Does D matter? The role of vitamin D in hair disorders and hair follicle cycling
- Hair follicle expression of 1,25-dihydroxyvitamin D3 receptors during the murine hair cycle
- Vitamin D and calcium regulation of epidermal wound healing
- Development and progression of alopecia in the vitamin D receptor null mouse
- Vitamin D and androgen receptor-targeted therapy for triple-negative breast cancer
- Targeted Expression of Human Vitamin D Receptor in the Skin Promotes the Initiation of the Postnatal Hair Follicle Cycle and Rescues the Alopecia in Vitamin D Receptor Null Mice
- Vitamin D‐dependent rickets type I and type II
- The Vitamin D Receptor Is Required for Activation of cWnt and Hedgehog Signaling in Keratinocytes
- Ligand-Dependent Actions of the Vitamin D Receptor Are Required for Activation of TGF-β Signaling during the Inflammatory Response to Cutaneous Injury
- The Vitamin D Receptor as Tumor Suppressor in Skin
- Molecular Genetic and Endocrine Mechanisms of Hair Growth
- The hair cycle and Vitamin D receptor
- Reciprocal role of vitamin D receptor on β-catenin regulated keratinocyte proliferation and differentiation
- Hereditary 1,25‐Dihydroxyvitamin D–Resistant Rickets Due to an Opal Mutation Causing Premature Termination of the Vitamin D Receptor
- Vitamin D Receptor Is Required for Proliferation, Migration, and Differentiation of Epidermal Stem Cells and Progeny during Cutaneous Wound Repair
- Vitamin D resistance
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Hereditary vitamin D-resistant rickets (HVDRR) owing to a heterozygous mutation in the vitamin D receptor
- Two New Unrelated Cases of Hereditary 1,25-Dihydroxyvitamin D-resistant Rickets with Alopecia resulting from the same Novel Nonsense Mutation in the Vitamin D Receptor Gene
- Vitamin D receptor polymorphisms and the polycystic ovary syndrome: A systematic review
- Correlation of vitamin D and vitamin D receptor expression in patients with alopecia areata: a clinical paradigm