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The lump on a woman's scalp was a rare, potentially cancerous tumor from the hair follicle, not a common cyst.

The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

A woman's rare ovarian tumor was treated with surgery, which stopped her symptoms and normalized her hormone levels.

A woman's male-like physical changes were caused by two rare ovarian conditions.

There might be a link between male pattern baldness and a higher risk of prostate cancer and testicular cancer.

A postmenopausal woman's virilization was caused by a rare ovarian tumor that was hard to detect but was successfully treated with surgery.

Ovarian steroid cell tumors should be considered in adults with hirsutism and high testosterone, with surgery as the main treatment.

Metformin can help diagnose and treat high testosterone levels in women, often due to insulin resistance rather than a tumor.

A woman's adrenal tumor disappeared after treatment with cyproterone acetate.

After chemotherapy for a gestational trophoblastic tumor, normal pregnancy rates are possible, but there's a slightly higher risk of the tumor reoccurring in future pregnancies.

The document concludes that understanding nail anatomy is key for diagnosing nail diseases, early signs of nail melanoma may allow for less aggressive treatment, and specific genetic mutations are important in thyroid cancer prognosis and treatment.

Ovarian stromal tumors are a rare cause of women developing male characteristics after menopause.

Woman with diabetes had hair loss due to rare ovarian tumor; surgery improved hair growth.

High levels of ornithine decarboxylase can cause tumors in mouse skin.

Overexpressing SSAT enzyme reduces prostate tumor growth in mice.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Increased ornithine decarboxylase makes normally tumor-resistant mice more sensitive to tumors.

Budesonide and N-acetylcysteine reduced tumors and alopecia in mice, regardless of FHIT gene status.

Metformin may help treat neuroendocrine tumors.

Progesterone byproduct 5αP stimulates mammary tumor growth, but finasteride can suppress it.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

Cutaneous Lymphadenoma is a unique skin tumor with specific protein markers and common gene mutations that may cause continuous cell growth.

Dutasteride may help shrink prostate cancer tumors.

The document concludes that identifying the specific cells where skin cancers begin is important for creating better prevention, detection, and treatment methods.

Giant solitary trichoepithelioma is a rare, usually non-cancerous hair follicle tumor that can sometimes turn into skin cancer.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Loss of TET2 increases the risk of skin and oral cancer.

Personalized treatment based on detailed tumor analysis successfully managed and reduced the patient's aggressive hair follicle cancer.

SOX4 is crucial for the development of melanoma.

Pygo2 is important for early growth and progression of intestinal tumors, and could be a target for treating cancers with certain mutations.