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A child with a rare vitamin D-resistant condition improved with treatment.

A rare genetic disease causes rickets and often hair loss in young children, which can improve with specific treatments.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

Two siblings stayed rickets-free for 14 years after stopping treatment.

A girl had rickets due to a gene mutation affecting vitamin D response.

Vitamin D-dependent rickets Type II causes bone problems and hair loss, and doesn't improve with Vitamin D treatment.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A boy's hair loss and bone issues improved with specific vitamin D treatment and calcium.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

A 3-year-old with vitamin D-resistant rickets and severe hair loss died despite treatment, highlighting the need for improved management of the condition.

Gene therapy with a vitamin D receptor gene improved hair growth in rats with a type of rickets-related baldness.

Two siblings with a rare type of rickets and hair loss had low vitamin D levels, which is not typical for their condition.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Intravenous and oral calcium effectively treated vitamin D dependent rickets type II, improving growth and bone health.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.