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Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

Somatostatin may help protect hair follicles from immune attacks.

Biotin supplements are unnecessary for most people but may help with certain conditions like biotin deficiency, brittle nails, and some hair loss.

Activating TLR3 improves the healing and immune properties of periodontal ligament stem cells.

Poor response to topical immunotherapy in alopecia areata patients is linked to impaired cell responses.

Valproic Acid could potentially be used to treat immune-related conditions due to its ability to modify immune cell functions.

Antroquinonol may help prevent skin depigmentation by suppressing certain immune cells.

A deficiency in the TTC7A gene causes immune problems, gut issues, and hair loss.

Wnt/beta-catenin signaling in the skin helps fat cell development during hair growth and repair.

Human body's immune cells are more common in the layer of fat just beneath the skin than in deeper fat layers.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

The secretome from mesenchymal stem cells shows promise for treating skin conditions and improving skin and hair health, but more research is needed.

Combination pharmacotherapy is generally more effective for treating keloids and hypertrophic scars.

Cell-based therapy using specific immune cells may help treat alopecia areata by promoting hair regrowth.

Exosomes could be key in treating skin conditions and healing wounds.

The document concludes that treatment for cutaneous T-cell lymphoma should be customized to each patient's disease stage, balancing benefits and side effects, with no cure but many patients living long lives.

People with alopecia areata have more Th17 cells and fewer Treg cells, which may be key to the condition's development.

Estrogen increases a growth factor in hair cells which might affect hair loss.

Changes to the Foxp3 protein affect how well regulatory T cells can control the immune system, which could help treat immune diseases and cancer.

A boy with alopecia regrew hair using a vitamin D cream after other treatments failed.

The S250C variant in a gene may cause autoimmunity and immunodeficiency by impairing protein function.

Tofacitinib was more effective than apremilast in treating hair loss in a mouse model of alopecia areata.

The congress highlighted new gene therapy techniques and cell transplantation methods for treating diseases.

Air pollution worsens skin diseases and aging by causing inflammation and oxidative stress.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

Zinc is effective for treating various skin conditions, including warts and acne.

Cepharanthine is a well-tolerated drug with multiple medical uses, including anti-inflammatory and anti-cancer properties.

Children with atopic dermatitis often have sleep problems, affecting their growth and behavior.

Animal models have helped understand hair loss from alopecia areata and find new treatments.

Lymphocytes, especially CD8+ T cells, play a key role in causing alopecia areata, and targeting them may lead to new treatments.