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The document concludes that the development of certain tumors is influenced by genetic background and that a specific gene modification can lead to tumor regression and reduced growth.

The gene Tfap2b is essential for creating a type of stem cell in zebrafish that can become different pigment cells.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Mice with human gene experienced hair loss when treated with DHT.

Advanced therapies like gene, cell, and tissue engineering show promise for hair regrowth in alopecia, but their safety and effectiveness need more verification.

Mutations in the HEPHL1 gene cause abnormal hair and cognitive issues.

Deleting the Far2 gene in mice causes sebaceous gland issues and patchy hair loss.

Disrupting the FGF5 gene in rabbits leads to longer hair by extending the hair growth phase.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

Removing a methyl group from the ITGAV gene speeds up bone formation in a specific type of bone disease model.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

The PON1 192R gene variant is linked to a higher risk of psoriasis and heart disease in Western Mexico.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

Colchicine can inhibit hair growth by affecting cell activity and protein expression in hair follicles.

Hair loss in certain young mice is linked to a specific gene and can be caused by lack of iron.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

Hemoglobin α acts as an antioxidant in skin cells, especially protecting hair follicle stem cells from UV damage.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

A woman with a unique syndrome similar to TRPS has a genetic change near the TRPS1 gene, affecting its regulation.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Caffeine can protect scalp hair follicles from damage caused by UV radiation.

Wnt ligands are crucial for hair growth and repair.

Researchers found that certain RNA circles in the brain are linked to disease risk, but their exact role in disease is still unknown.

The review suggests thorough evaluation and genetic testing for proper diagnosis and treatment of Chrousos syndrome.

Non-coding RNAs play key roles in the hair growth cycle of Angora rabbits.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

NFIC helps rat dental cells grow and turn into bone-like cells.

Specific RNA patterns are linked to alopecia areata.