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Nestin-expressing cells turn into a specific type of skin cell in hair follicles during development and in adults.

Nestin marks cells that can become a specific type of skin cell in hair follicles of both developing and adult mice.

Nestin identifies specific progenitor cells in hair follicles that can become outer root sheath cells.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

Keratin 79 marks a new group of cells that are key for creating and repairing the hair follicle's structure.

Certain genes are more active in baby scalp cells and can help grow hair when added to adult mouse skin cells.

Different types of stem cells in hair follicles play unique roles in wound healing and hair growth, with some stem cells not originating from existing hair follicles but from non-hair follicle cells. WNT signaling and the Lhx2 factor are key in creating new hair follicles.

A specific DNA region is crucial for Foxn1 gene expression in thymus cells but not in hair follicles.

Wnt signaling is crucial for pigmented hair regeneration by controlling stem cell activation and differentiation.

Hair follicle stem cells are a practical and ethical option for nerve repair in regenerative medicine.

Phosphatidic acid may help hair grow by affecting cell growth pathways.

Scientists have found a way to create hair follicles from skin cells of newborn mice, which can grow and cycle naturally when injected into adult mouse skin.

A pathway helps maintain long telomeres in both stem and cancer cells.

Melanocyte precursors in human fetal skin follow a specific migration pattern and some remain in the skin's deeper layers.

After skin injury, adult mice can grow new hair follicles, and this process can be increased or stopped by manipulating Wnt signals.

Eyelid cells share signaling components but differ in pathway activity.

Hair and nail cells share similar proteins, indicating a common differentiation pathway.

The document concludes that the skin's immune system is complex, involving interactions with hair follicles, nerves, and microbes, and can protect or cause disease, offering targets for new treatments.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

Certain proteins, Lgr5 and Lgr6, are important markers of adult stem cells and are involved in tissue repair and cancer development.

Removing Mediator 1 from certain mouse cells causes teeth to grow hair instead of enamel.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Grafted rodent and human cells can regenerate hair follicles, but efficiency decreases with age.

Certain signals are important for reducing specific chemical markers on hair follicle stem cells during rest periods, which is necessary for healthy hair growth.

Wnt proteins from certain skin cells are crucial for normal hair growth and renewal.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Drosha and Dicer are essential for hair follicle health and preventing DNA damage in skin cells.

HPV16 oncogenes disrupt the normal activity of hair follicle stem cells.

Different cell types work together to repair skin, and targeting them may improve healing and reduce scarring.

EDA and EDAR are important for hair follicle development in cashmere goats and affect other related genes.