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A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

Older mice have stiffer skin with less elasticity due to changes in collagen and skin structure, affecting aging and hair loss.

Human sebaceous glands can grow back in skin grafts on mice and work like normal human glands.

Ovariectomized mice mimic postmenopausal hair loss, and estradiol helps maintain hair density.

Younger mice's hair-follicle stem cells are better at turning into heart cells than older mice's.

A new model using mice with human hair follicles helps better understand hair loss from chemotherapy.

The "Punch Assay" can regenerate hair follicles efficiently in mice and has potential for human hair regeneration.

Herbal extracts promoted hair growth similar to minoxidil in mice.

Animal models, especially mice, are essential for advancing hair loss research and treatment.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Researchers successfully used nude mice to study human hair growth, which could help with future hair research.

Spiny mice regenerate skin better than laboratory mice due to larger hair bulges, more stem cells, and different collagen ratios.

Researchers created a 3D-printed skin model that grew human hair when grafted onto mice by improving blood supply to the grafts.

Older mice healed wounds better but lost more weight and might have weaker immune systems afterward.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

Hair stem cells were tracked in mice using a special imaging technique, showing that it's possible to monitor hair growth this way.

LhGH promotes hair growth and prevents hair loss in mice.

Activating TERT in mice skin boosts hair growth by waking up hair follicle stem cells.

Hair color production in mice is closely linked to the hair growth phase and may also influence hair growth itself.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Hair patterns in mice are controlled by both a global system dependent on Fz6 and a local self-organizing system.

Different body areas in mice produce different hair types due to interactions between skin layers.

The p53 protein helps control hair follicle shrinking by promoting cell death in mice.

Healthy mitochondria in skin cells are essential for proper hair growth and skin cell interaction in mice.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Cedrol from Platycladus orientalis leaves may promote hair growth effectively, especially in female mice.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Human scalp hair on mice can effectively monitor mercury exposure.

Stress in mice delays hair growth and treatments blocking substance P can partly reverse this effect.