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Deleting the CRIF1 gene in mice disrupts skin and hair formation, certain proteins affect hair growth, a new compound may improve skin and hair health, blood cell-derived stem cells can create skin-like structures, and hair follicle stem cells come from embryonic cells needing specific signals for development.

Ishige sinicola extract helps bone-building cells grow and mature, which could aid in treating osteoporosis.

Antibody treatments show promise for hair loss but need more research.

Hair follicles supply a crucial brain development protein to the brain via platelets.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

The vitamin D receptor can work without its usual activating molecule.

Vitamin D receptor helps control hair growth genes in skin cells.

Scientists found new nonsteroidal compounds that can act like natural male hormones and might help treat male fertility and hormone issues.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Targeting the Smoothened receptor shows promise for treating certain cancers.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Notch1 signaling is crucial for improving wound healing and skin regeneration by affecting stem cell behavior.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

TLR2 is important for hair growth and can be targeted to treat hair loss.

The androgen receptor's shape-changing ability helps it function but can lead to cancer treatment resistance.

Vitamin D receptor is crucial for skin wound healing.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

The JAK/STAT pathway is important in cell processes and disease, and JAK inhibitors are promising for treating related conditions.

Fibroblast growth factor receptor signaling controls cell development and repair, and its malfunction can cause disorders and cancer, but it also offers potential for targeted therapies.

EGF and KGF signalling prevent hair follicle formation and promote skin cell development in mice.

The research found how GPCR Class A Rhodopsin receptors are related and suggested possible substances they interact with.

Older skin has higher cancer risk due to inflammation and stem cell issues.

Vitamin D and its receptor may help prevent skin cancer.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Vitamin D receptor may help protect against UV-induced skin cancer.

Androgens prevent hair growth by changing Wnt signals in cells.

The Wnt/β-catenin pathway can activate melanocyte stem cells and may help regenerate hair follicles.

NcoA4 may have roles beyond helping control gene activity, possibly affecting cell behavior and stability.