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Progesterone and its byproducts control a specific receptor in the brain independently of progesterone receptors, affecting conditions related to the menstrual cycle.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

Blocking DGAT1 reduces oil gland size in mice and dogs, but only mice experience hair loss.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Genomic research can help improve the quality and production of natural fibers in animals.

Blocking SEPT4 might help heal wounds and regrow hair faster.

TLR3 activation helps improve skin and hair follicle healing in mice.

PTHrP is important for bone formation and may be targeted for osteoporosis treatment and longevity therapies.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

15-lipoxygenase helps keep skin healthy by reducing inflammation.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

Pannexin 3 helps skin and hair growth by controlling a protein called Epiprofin.

Allopregnanolone is needed for certain brain processing issues caused by D1 dopamine receptor activation.

The HIF-2α/ARNT complex is important for hair follicle development by controlling cell growth.

Animal models, especially mice, are essential for advancing hair loss research and treatment.

Swiss Holstein cattle with curly, short hair carry genes from the Simmental breed.

Dicer is crucial for hair growth in mice.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Twist2 is essential for scarless skin healing and hair growth in mouse fetuses.

Twist2 is essential for proper skin healing and hair growth in developing mice.

Removing the ATR gene in adult mice causes rapid aging and stem cell loss.

Blocking the enzyme 5α-reductase can shrink the prostate and help treat enlarged prostate issues.

Blood vessels and specific genes help turn cartilage into bone when bones heal.

Gene therapy with the Math1 gene helped regenerate balance-related cells and improve balance in mice.

The document concludes that assessing hair follicle damage due to cyclophosphamide in mice involves analyzing structural changes and suggests a scoring system for standardized evaluation.

The p75 neurotrophin receptor is important for hair follicle regression by controlling cell death.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.