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Women with excessive hair growth or polycystic ovary disease may more often carry a gene variant for 21 hydroxylase deficiency.

Melanogenesis-related proteins may trigger immune responses in alopecia areata patients.

Alopecia areata can be transferred through stem cell transplants from affected siblings.

EGCG may help treat alopecia areata by blocking certain immune responses and reducing specific harmful immune cells.

Lichen planus is a skin condition that can resolve on its own, is linked to hepatitis C, and increases the risk of skin cancer.

Some cancer treatments can cause abnormal fine hair growth.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

Alopecia areata, a common autoimmune hair loss condition, often runs in families.

The document concludes that ongoing research using animal models is crucial for better understanding and treating Alopecia Areata.

Certain HLA class II alleles increase or decrease the risk of alopecia areata.

Lichen Planopilaris is a hair loss condition best treated early with various medications, including hydroxychloroquine, to prevent permanent baldness.

The document concludes that Cutaneous Lupus Erythematosus has different forms, is influenced by genetic and environmental factors, and can be treated with various medications, but more targeted therapies are needed.

Half of people with Alopecia Areata may see hair regrowth within a year without treatment, but recovery is unpredictable.

Lichen planus pigmentosus causes dark skin patches and is treated by avoiding triggers and using anti-inflammatory medications.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Alopecia Areata has no guaranteed treatment for hair regrowth, but options like corticosteroids and minoxidil are used, with future research focusing on genetic and immune therapies.

Despite progress in treatment, the exact cause of Alopecia areata is still unknown.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Genetic factors alone might not cause pemphigus vulgaris; other factors like birth complications and puberty may trigger it.

HLA-DRB5 and other genes may be linked to alopecia universalis.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

Alopecia areata has a complex genetic basis that was not fully understood as of 2001.

The C-allele and CC-genotype in the PTPN22 gene lower the risk of alopecia areata.

Some alternative therapies for vitiligo show promise but need more research.

Obesity in the U.S. is largely influenced by diet and exercise, certain medications can improve heart procedures and hair growth, and major teaching hospitals have better patient outcomes.

Terbinafine is more effective than itraconazole for toenail fungus, especially in older patients, and debridement improves its effectiveness.

Herpes gestationis is linked to certain antigens, atopic eczema affects T cell populations and may be eased by breastfeeding, higher doses of anti-androgen treatment can improve androgenic alopecia, topical minoxidil increases hair thickness, long-term methotrexate therapy can cause liver fibrosis in psoriasis patients, and Lichen Sclerosus et Atrophicus patients aren't at higher risk for autoimmune disorders.

Mutations in the KRT85 gene cause hair and nail problems.

Busulfan/cyclophosphamide and total bone irradiation are equally effective for AML transplants.