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Washing permed hair after using thioglycolic acid helps reform strong bonds, making hair stronger.

Hair's strength and flexibility come from its protein structure and molecular interactions.

Hair growth is controlled by specific gene clusters and proteins, and cysteine affects hair gene expression in sheep.

Changes in keratin make hair follicles stiffer.

George Ernest Rogers was a notable scientist who made important discoveries about hair and wool proteins.

Human hair keratin fibers have a detailed nano-scale structure that changes with different conditions.

CMADK reduces hair damage from bleaching and permanent waving.

Trichohyalin is a protein in hair follicles that helps form hair filaments.

Permanent wave treatment with thioglycolic acid changes hair structure by altering disulfide bonds.

CIP/KIP proteins help stop cell division and support hair growth.

Keratin structure in hair is stable at pH 5-6 but disrupts between pH 6-7.

Human hair keratins K85 and K35 create unique filament patterns important for early hair formation.

Curved hair can develop when hair cells merge abnormally during growth.

Hair curvature in Japanese people is linked to specific cell types and filament arrangements in the hair cortex.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

The model shows that filament flexibility and amino acid differences affect how fast intermediate filament proteins assemble.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

The document concludes that specific cells are essential for hair growth and more research is needed to understand how to maintain their hair-inducing properties.

Two specific hair keratin genes are active during hair growth and decline as hair transitions to rest.

Hair's structure and properties change with pH; acidic pH maintains strength and less swelling, while alkaline pH increases water content and swelling.

The antibody created from BCC tissues reacts similarly to both BCC and hair follicles, suggesting BCC may come from hair follicle cells.

Calreticulin has roles in healing, immune response, and disease beyond its known functions in the endoplasmic reticulum.

Fat cells help recruit healing cells and build skin structure during wound healing.

Fat-derived stem cells show promise for skin repair and reducing aging signs but need more research for consistent results.

The conclusion is that tissue structure is key for stem cell communication and maintaining healthy tissues.

The conclusion is that hair growth can be improved by activating hair cycles, changing the surrounding environment, healing wounds to create new hair follicles, and using stem cell technology.

Skin problems can be caused or worsened by physical forces and pressure on the skin.

Notch-related genes play a key role in the development and cycling of hair follicles.

Melatonin helps Cashmere goats grow more hair by affecting certain genes and cell pathways.