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Finasteride affects offspring's antioxidant enzymes in epididymis, possibly disrupting sperm maturation.

Advancements in hair follicle culture have led to better understanding and potential treatments for hair loss.

Blocking the Mitochondrial Pyruvate Carrier causes stress in hair follicles, which can be reduced by an ISR inhibitor.

The mTOR signaling pathway is crucial for hair health and targeting it may lead to new hair loss treatments.

Androgenic alopecia causes hair follicle degradation and skin restructuring, but some hair elements remain.

MOF controls skin development by regulating genes for mitochondria and cilia.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Wnt/beta-catenin signaling in the skin helps fat cell development during hair growth and repair.

The document concludes that understanding the sebaceous gland's development and function is key to addressing related skin diseases and aging effects.

Cancer can hijack the body's cell repair system to promote tumor growth, and targeting this process may improve cancer treatments.

Hair follicle stem cells are a promising source for tissue repair and treating skin or hair diseases.

Ludwig pattern hair loss in women results from varying sensitivity in hair follicles, causing fewer visible hairs.

Fat cells are important for tissue repair and stem cell support in various body parts.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

Skin organoids are a promising new model for studying human skin development and testing treatments.

Adding human fat-derived stem cells to hair follicle grafts greatly increases hair growth.

Melanocytes are diverse cells important for pigmentation and skin health, influenced by genetics and environment.

The proteins transthyretin and megalin are more present in the growth phase of hair, suggesting they might affect hair health and growth.

Melanocytes produce melanin; their defects cause vitiligo and hair graying, with treatments available for vitiligo.

Deleting the CRIF1 gene in mice disrupts skin and hair formation, certain proteins affect hair growth, a new compound may improve skin and hair health, blood cell-derived stem cells can create skin-like structures, and hair follicle stem cells come from embryonic cells needing specific signals for development.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

Scientists created a tiny, 3D model of a hair follicle that grows and acts like a real one.

Cholesterol sulfate buildup due to a genetic mutation disrupts the skin barrier, leading to the scaling skin seen in X-linked ichthyosis.

Mice without collagen VI have slower hair growth normally but faster regrowth after injury.

A special environment is needed to fully activate sleeping stem cells.

Bioluminescence imaging can track hair follicle cells and help study hair regrowth.

Different ectodermal organs like hair and feathers regenerate differently, with specific stem cells and signals involved in their growth and response to the environment.

Macrophages are more involved in Lichen planopilaris than in Frontal fibrosing alopecia.

Researchers improved a method to study individual cells in newborn mouse skin and found a way to assess the severity of a skin condition in humans.