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Dihydrotestosterone can be made from dehydroepiandrosterone in skin cells without needing testosterone.

The conclusion is that accurately identifying the cause of high androgen levels in women with PCOS is crucial and requires specific tests.

Muscles can make their own androgens, which may help muscle growth.

Epitestosterone may act as a weak antiandrogen and can inhibit an enzyme involved in testosterone metabolism.

The exact cause of increased 5α-reductase activity leading to hirsutism in women is still unknown.

Some hormone-related drugs may protect brain cells in Parkinson's disease differently in men and women.

Sex hormone treatments can increase muscle mass in older adults but have inconsistent effects on muscle function and may carry cardiovascular risks.

The document explains hair biology, the causes of hair loss, and reviews various hair loss treatments.

Minoxidil and caffeine in transfersomes improve hair growth treatment.

Dihydrotestosterone increases certain inflammatory signals in skin cells, potentially contributing to acne.

Finasteride effectively treats male hair loss, improving growth and density.

Women with female pattern hair loss have higher levels of certain androgens, suggesting increased androgen exposure to hair follicles.

Early adrenal gland maturation in young children can be normal, but other serious conditions should be ruled out first.

Androgens cause oil-producing skin cells with androgen receptors to mature and produce more oil.

Citrullus colocynthis fruit extract may effectively treat testosterone-related hair loss, similar to finasteride.

5-alpha-reductase inhibitors may cause a low incidence of erectile dysfunction that decreases over time.

Testosterone and dihydrotestosterone play a role in women's health issues like excess hair and baldness, and treatments blocking these hormones may help.

Estradiol and castration reduced prostate cancer development in rats when applied at early stages, but were ineffective after cancer was established.

Certain HSD3B1 gene types are linked to worse prostate cancer outcomes and affect treatment response and other health conditions.

The conclusion is that future hair loss treatments should target the root causes of hair thinning, not just promote hair growth.

The model suggests that scalp tension could lead to hair loss, with factors like blood vessel hardening, enlarged oil glands, and poor microcirculation also playing a role. It also hints at a possible link between skull shape and baldness pattern.

Men with common hair loss may have a higher risk of heart disease and diabetes, and should be checked for these conditions.

Androgen receptor signaling causes early aging of cells important for hair growth by damaging their DNA.

Analyzing hair with this method can help understand and monitor scalp conditions and treatment effects.

Human keratinocytes do not naturally respond to androgens.

SHBG binding properties are unchanged in male liver cirrhosis patients.

Women with excessive male-pattern hair growth should get a full hormone check-up to find and treat any underlying issues, considering both medical and emotional aspects.

Hair disorders are caused by a complex mix of biology, genetics, hormones, and environmental factors, affecting hair growth and leading to conditions like alopecia.

Lipocalin-2 levels are higher in women with PCOS and may indicate insulin resistance.

New treatments for hair loss show promise, especially finasteride for men and a stronger minoxidil formula.