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The STRIPAK complex is crucial for skin cell organization and creating a functional skin barrier.

The hexosamine pathway helps maintain healthy skin by affecting the skin's structure and possibly increasing hair follicle stem cells.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

New drugs for Alzheimer's and rheumatoid arthritis advanced, a Zika vaccine is in development, and there were business deals in anesthesia and oncology.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

The hexosamine pathway helps protect skin cells from stress and may improve skin and hair health.

Pdgfα signaling helps maintain fat cells in the skin and is important for hair growth, wound healing, and fighting infections.

Valentina Greco's research shows that the environment around hair follicle stem cells is more crucial for regeneration than the stem cells themselves.

The right amount of retinoic acid is essential for normal hair growth and development.

Lack of Crif1 in hair follicle stem cells slows down hair growth in mice.

Collagen VII helps skin heal and stay strong, sirolimus may lower skin cancer risk in kidney transplant patients, high-molecular-mass hyaluronan helps naked mole rats resist cancer, dermal γδ T cells aid in hair growth in rodents, and overexpression of IL-33 in mouse skin causes itchiness, offering a model for studying allergic inflammation treatments.

Blood vessels and specific genes help turn cartilage into bone when bones heal.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Mutant mice help researchers understand hair growth and related genetic factors.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

The document concludes that careful design of genetic fate mapping experiments is crucial for accurate cell lineage tracing in mice.

ARL15 is important for fat cell development and the release of the hormone adiponectin.

New treatments for diabetes, central nervous system repair, and cartilage injury were found, and a way to create functional hair follicles from stem cells was developed.

Understanding where cancer cells come from helps create better prevention and treatment methods.

Targeting androgen receptors could be a promising way to treat skin disorders with fewer side effects.

MIM is crucial for hair follicle formation and regeneration by controlling cilia formation and hedgehog signaling through its interaction with Cortactin and Src.

mTOR signaling helps activate hair stem cells by balancing out the suppression caused by BMP during hair growth.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

TGFβ's manipulation of inflammation and immune cells affects cancer spread, suggesting new treatment strategies and biomarkers.

Mice can correct hair follicle orientation without certain genes, but proper overall alignment needs those genes.

The research suggests that a specific skin gene can be controlled by signals within and between cells and is wrongly activated in certain skin diseases.

Using polyethylenimine-DNA to deliver the hTERT gene can stimulate hair growth and may be useful in treating hair loss, but there could be potential cancer risks.

Twist1 helps maintain important features of cells crucial for hair growth by working with Tcf4 and β-catenin.

Runx genes are important for stem cell regulation and their roles in aging and disease need more research.

Spironolactone might lower the chance of getting rosacea.