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Severe hair loss (androgenetic alopecia) is linked to higher risk of heart disease (cardiovascular atherosclerosis).

Some heart drugs show promise for other conditions, but more research is needed to confirm their effectiveness and safety.

Vitamin D and estrogen may help protect heart and kidney health, and maintaining sufficient vitamin D levels could be especially beneficial for African Americans, postmenopausal women, and people with chronic kidney disease.

Human hair protein modifications could potentially indicate heart disease risk.

Male hormones may play a role in the development of heart disease, and more research is needed to understand their effects.

Balding men have higher heart disease risk.

Some skin conditions may increase the risk of heart disease, but are not yet included in cardiovascular prevention guidelines.

Some medications and supplements can cause serious side effects, including hair loss, heart rhythm problems, liver injury, and other health issues.

Understanding and tracking our body's natural daily rhythms could help improve heart health.

High fasting insulin levels in women with PCOS are linked to a higher risk of heart and metabolic problems.

Men's age, urinary symptoms, and heart health risks create different types of erectile dysfunction.

DHT may increase the risk of heart disease and death in elderly men.

Tacrolimus causes fewer acute rejections than cyclosporin A in kidney transplants but doesn't necessarily improve kidney function after one year; cardiovascular risks and side effects vary between the two drugs.

Some skin conditions may increase the risk of heart disease, and understanding their connection could lead to better treatments.

JAK-inhibitors for alopecia areata are generally safe with mostly mild side effects and a low rate of treatment withdrawal.

Testosterone therapy in transgender men has both desired effects like increased muscle mass and potential health risks such as higher cardiovascular risk.

Older obese women with PCOS have higher cardiovascular and metabolic risks despite lower androgen levels.

Testosterone therapy for postmenopausal women appears safe and may protect against heart disease, but requires constant monitoring and more research for long-term effects.

5-alpha reductase inhibitors can cause sexual, neurologic, endocrine, and cardiovascular side effects, but these are rare and usually stop after ending treatment.

PCOS in lean women is a serious health condition with implications beyond fertility, affecting metabolism and increasing cardiovascular disease risk.

Skin diseases like psoriasis, lupus, and others can increase heart disease risk due to factors like chronic inflammation and genetic susceptibility.

Hair loss in adults is linked to heart disease risk factors like unhealthy blood fats, lack of exercise, and alcohol use. It's also slightly more common in people with metabolic syndrome. As people age, hair loss gets worse. In men, severe hair loss is tied to high blood pressure and a larger waist. In women, it's tied to higher body weight. People with hair loss should be checked for unhealthy blood fats and advised to avoid alcohol and inactivity.

Testosterone is linked to cardiovascular risk factors and stroke, but its exact role is unclear.

People with androgenetic alopecia have a higher risk of heart disease.

Sex hormone treatments can increase muscle mass in older adults but have inconsistent effects on muscle function and may carry cardiovascular risks.

JAK inhibitors for alopecia areata are linked to minor side effects like headache and acne, but not to an increased risk of serious adverse events.

High androgen levels in young women with PCOS are linked to higher blood pressure.

Severe vertex pattern hair loss may indicate a higher risk for artery plaque buildup.

Small changes in hormones can significantly impact health, showing the importance of sensitive testing for chemicals that disrupt hormones.

Rosuvastatin improves lipid levels, reduces inflammation, but worsens insulin sensitivity in men with early-onset hair loss.