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Intravenous and oral calcium effectively treated vitamin D dependent rickets type II, improving growth and bone health.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

A child with a rare vitamin D-resistant condition improved with treatment.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Vitamin D receptor interacts with certain dietary components to help prevent diseases and regulate hair growth.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Two siblings stayed rickets-free for 14 years after stopping treatment.

Vitamin D is crucial for bone health and preventing serious diseases.

Six genetic conditions are often linked to complete scalp hair loss in children.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Stem cell therapy, particularly using certain types of cells, shows promise for treating hair loss by stimulating hair growth and development, but more extensive trials are needed to confirm these findings.

The document corrects errors in previous articles, including a reversed term, updated subject weights, hair count data, and reference numbering.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Putting calcipotriol ointment on the skin once doesn't change calcium levels in the body right away.

ACTH may be an effective first-line treatment for acute calcium pyrophosphate crystal arthritis.

The document concludes that various skin conditions have specific treatments and that adequate calcium intake may prevent osteoporosis.

A study found that minoxidil can damage the hearts of dogs and pigs, cautioning its use in humans.