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Skin and hair can regenerate after injury due to changes in gene activity, with potential links to how cancer spreads. Future research should focus on how new hair follicles form and the processes that trigger their creation.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Thyroid hormones affect skin texture, hair and nail growth, and can cause skin diseases related to thyroid problems.

Minoxidil promotes hair regrowth in early baldness stages and prevents baldness in non-bald scalps.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

Scientists created early-stage hair follicles from human skin cells, which could help treat baldness and study hair growth.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

The paper concludes that understanding melanocyte development can help in insights into skin diseases and melanoma diversity.

ATP increases melanin production in skin after UV exposure, with the P2X7 receptor being crucial for this process.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

The May issue discussed publishing agreements and four studies on cholesterol in hair, cancer cell changes, hormones in the uterus, and skin protein effects.

The document concluded that stem cells are crucial for skin repair, regeneration, and may help in developing advanced skin substitutes.

Apoptosis is essential for human skin development and forming a functional epidermis.

Melatonin stimulates the skin components of ram's scrotum during their non-breeding season.

Adding hyaluronic acid helps create larger artificial hair follicles in the lab.

OCT binds strongly to hair sheath cells and may affect skin and hair growth with fewer side effects than vitamin D3.

The cornified envelope is crucial for skin's barrier function and involves key proteins and genetic factors.

Bacteria can help skin regenerate through a process called IL-1β signaling.

SARS-CoV-2 might infect and multiply in skin tissue, possibly aiding in its transmission.

Radiation therapy damages skin structure and immune function, causing inflammation and potential hair loss.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Human foreskin does not show aging or reduced cell growth after radiation, and H2A.J is not a good marker for radiation-induced aging.

The skin acts like an endocrine organ, producing hormones that affect various body functions and skin health, and understanding this can lead to new treatments.

A new ethical skin model using stem cells offers a reliable alternative for dermatological research.

Skin stem cells are promising for healing wounds and skin regeneration due to their accessibility and regenerative abilities.

IL-9 increases skin cell movement but decreases their ability to invade, and this effect is controlled by cell contractility, not by MMPs.

The enzyme 25 Hydroxyvitamin D 1 α-Hydroxylase is essential for healthy skin and recovery after skin damage.

Dsg1 is essential for maintaining a healthy skin barrier in mice.

Epidermal stem cells are diverse and vary in activity, playing key roles in skin maintenance and repair.

TCDD speeds up skin barrier formation by increasing certain gene expressions.