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The conclusion is that standardizing testosterone measurement methods is essential for better diagnosis accuracy in women.

Both finasteride and GnRH agonist treatments reduced hair growth in women with idiopathic hirsutism, but GnRH agonist was more effective.

About 19.4% of Iranian women in the study have Polycystic Ovary Syndrome, with the most common type involving irregular periods and high male hormone levels, but their heart and metabolic health is similar to women without the condition.

Prostaglandin D2 increases testosterone production in skin cells through a process involving reactive oxygen species, and antioxidants may help treat hair loss.

Blood AMH levels are higher in women with PCOS than in those with other similar conditions.

The treatment significantly reduced hair growth and male hormone levels in hirsute women.

Certain adrenal hormones can strongly stimulate oil gland growth in hamster skin, similar to male hormones.

Simvastatin may help manage symptoms in women with non-classic congenital adrenal hyperplasia by lowering cholesterol and certain hormone levels.

LC-MS/MS is more reliable than immunoassays for diagnosing 21-hydroxylase deficiency.

Metformin improved menstrual cycle regularity and signs of hyperandrogenism in adolescent girls with type 1 diabetes but did not improve blood sugar control.

Chinese women with PCOS have higher androgen levels, which change with age and weight.

Genetic defects in testosterone production can cause hormonal and developmental disorders, and more research is needed to understand androgen regulation and develop safer treatments.

Untreated congenital adrenal hyperplasia can lead to complications like stroke and needs glucocorticoid treatment.

Flutamide and finasteride reduced sex hormones in pregnant hyenas, but increased them in males.

Finasteride, when taken daily, lowers dihydrotestosterone levels but doesn't noticeably affect adrenal steroid production, except by blocking the 5 alpha-reductase enzyme.

The treatment temporarily improved symptoms of hyperandrogenism in adolescents.

Cyproterone acetate implants were more effective at reducing facial hair and improving skin in severe hirsutism than oral treatment.

The treatment reduced hair growth and testosterone in women with excess hair and had some effects on stress responses.

Cyclosporine A therapy changes sex hormone levels differently in pre-menopausal and post-menopausal women with primary biliary cirrhosis.

3α, 17β-androstanediol-glucuronide is not a useful marker for androgen excess but may help monitor certain treatments.

Most women with polycystic ovary syndrome (PCOS) have menstrual issues, infertility, acne, and excessive body hair, with the most common type being the non-androgenic phenotype.

Women with premature ovarian insufficiency have a worse cardiovascular risk profile than premenopausal women.

Women with PCOS and polycystic ovaries have higher male hormone levels and more insulin resistance, especially if they are overweight.

The chapter explains the causes of excessive hair growth and masculinization in women and how to measure hormone levels related to these conditions.

The treatment effectively reduced hair growth and was safe for patients with PCOS, but it needs better bleeding control.

Higher androgen levels during puberty are linked to shorter adult height in boys with Silver-Russell syndrome.

A woman's mature cystic teratoma caused her virilization by producing testosterone.

Some supplements and hormones can increase hair loss by raising DHT levels.

A rare ovarian tumor caused high testosterone in a postmenopausal woman, resolved by surgery.

Rare ovarian tumors can cause hyperandrogenism, even if imaging appears normal.