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Vitamin D is important for skin cell growth and health, and its active form and receptor play key roles in skin and hair processes.

The skin produces and uses vitamin D for bone health, cell growth, and immune function.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Vitamin D is important for more than just bone health.

The skin acts like an endocrine organ, producing hormones that affect various body functions and skin health, and understanding this can lead to new treatments.

Low vitamin D levels are linked to increased risk of male pattern baldness.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Patients with Discoid Lupus Erythematosus have lower vitamin D and antioxidant levels and higher oxidative stress.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

Redheaded people may have evolved to efficiently make vitamin D in areas with less sunlight.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Effective treatments for scalp psoriasis include glucocorticosteroids, Vitamin D3 analogs, and combination therapies like calcipotriol and betamethasone dipropionate.

Vitamin D receptor is crucial for skin wound healing.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Balanced vitamin D levels are crucial to delay aging and prevent related diseases.

The document concludes that Borrelia afzelii causes a skin condition in France, a gene is linked to hair loss in Caucasian women, and various genetic mutations affect skin diseases.

Two siblings with gray patch tinea capitis were successfully treated after identifying risk factors.

Vitamin D supplements may reduce hair-pulling in people with Trichotillomania.

Scalp cooling is the only FDA-approved method to prevent hair loss from chemotherapy, but other treatments like minoxidil and PRP are being tested.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

People with alopecia areata often have lower vitamin D levels and more inflammation, suggesting vitamin D might be involved in the condition.

Low ferritin levels might be linked to telogen effluvium, but vitamin D levels are not.

Patients with myotonic dystrophy often have skin problems that suggest early aging and vitamin D issues, and the severity of these problems is linked to their genetic condition and vitamin D levels.

Many people in Majmaah city, Saudi Arabia, don't know much about vitamin D deficiency and don't practice good vitamin D habits, with few linking it to hair loss.