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Women with female pattern hair loss have higher levels of certain androgens, suggesting increased androgen exposure to hair follicles.

A three-month treatment with a GnRH agonist significantly lowered androgen levels and 5α-reductase activity in men with benign prostatic hyperplasia.

Certain inhibitors can potentially treat prostate cancer and other hormone-dependent conditions by controlling sex hormone levels in cells.

Topical spironolactone cream doesn't cause hormone-related side effects in the body.

The enzymes 5α-reductase and 3α/β-hydroxysteroid oxidoreductase help create brain-active substances from progesterone and testosterone, which could be used for treatment, but more research is needed to ensure their safety and effectiveness.

Flutamide, an antiandrogen, has minimal impact on female rat endocrine systems and does not significantly change their reproductive cycles.

Dehydroepiandrosterone (DHEA) is a prohormone important for producing sex steroids and has potential health benefits.

Blocking 5α-reductase can harm memory and brain structure, and increase harmful brain changes in male mice used for Alzheimer's disease research.

Women with low SHBG levels and a high 3a-diol G to SHBG ratio are likely to experience female pattern baldness, possibly due to a slight excess of androgens affecting sensitive hair bulbs.

Cocaine impairs male sexual behavior and alters testosterone metabolism in the brain.

Women with hyperandrogenism have higher androgen levels and lower SHBG, which may contribute to conditions like excessive hair growth and early puberty.

Finasteride affects brain and blood steroids, causing lasting sexual and emotional side effects.

Lack or blocking of SRD5a, a key component in hormone creation, can lead to conditions like pseudohermaphrodism and affect hair growth, bone mass, muscle strength, and reproductive health. More research is needed on its regulation from fertilization to adulthood.

Spironolactone reduces the enzyme activity that converts testosterone to DHT, helping treat excessive hair growth in women.

Finasteride treatment changes brain steroid levels and receptors, affecting brain function even after stopping treatment.

Testosterone and some of its forms can strongly stimulate oil gland growth in skin.

Androgen conjugates might be better indicators of skin sensitivity to hormones in women with excessive hair growth.

Androgens like testosterone are important hormones for both men and women, made differently in each sex and affecting the body by regulating genes and quick interactions with cell components.

Male rats have androgen receptors in their liver, which are different from proteins that bind estrogen.

Low-dose finasteride reduces excessive hair growth in teenage girls safely and affordably.

Male and female bodies respond differently to stress, influenced by hormones and development stages, with implications for stress-related diseases.

Androgens play a role in female sexual function, and testosterone therapy can help women with low sexual desire, but more research is needed on treatments and long-term safety.

Lower levels of certain brain chemicals are linked to worse PTSD symptoms in men.

An extract from Quercus acutissima bark was found to reduce sebum production and block an enzyme linked to acne.

Proscar (finasteride) blocks 5α-reductase in sea urchin ovaries and testes, suggesting potential treatment for androgen-related conditions.

Excessive hair growth in women often has no known cause and is not linked to race or other hormonal symptoms.

Certain genetic variations in the AR and ERβ genes can affect androgen levels in women.

Finasteride, CPA, and flutamide are all equally effective in reducing excessive hair growth in women.

People with hair loss may have different levels of certain hormones due to changes in hormone processing.

The document concludes that combination therapy is most effective for treating excessive hair growth in women with idiopathic hirsutism, and more research is needed to understand the condition.