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Blocking enzymes that help the virus enter cells could be a promising way to treat COVID-19.

COVID-19 can cause different skin symptoms that may help with early diagnosis and show how severe the disease is.

Skin problems in COVID-19 patients are rare and may be due to the body's complex immune response or blood clotting issues.

SARS-CoV-2, the virus causing COVID-19, can affect all endocrine organs and systems, altering their function and potentially leading to disorders. Factors like diabetes and obesity increase infection risk and severity. Understanding these effects is key for effective treatment.

Sex hormones may affect COVID-19 severity, with men often faring worse, and targeting related pathways could offer treatment options.

Androgen deprivation therapy might be better for preventing COVID-19 than treating it.

Men's sensitivity to male hormones might affect how severe COVID-19 gets for them.

Men have worse COVID-19 outcomes than women due to genetic and hormonal differences.

Betacoronaviruses, like COVID-19, may cause hormone system dysfunction and affect disease susceptibility and severity.

Men, especially older ones with health issues like prostate cancer, may have worse COVID-19 outcomes and could benefit from therapies targeting male hormones.

Steroid sex hormones activate matriptase in prostate cancer cells but not in breast cancer cells.

Men generally have more severe COVID-19 cases and higher death rates than women due to biological differences.

Mesenchymal stromal cells may help treat severe COVID-19, but more research is needed to confirm their effectiveness.

Traditional Chinese medicinal foods may help manage long-term post-COVID symptoms.

NF-κB signaling is crucial in many diseases and can be targeted for new treatments.

Testosterone may influence COVID-19 severity and outcomes.

Male hormones like testosterone may make COVID-19 worse, and testing for sensitivity to these hormones could help predict how severe a patient's symptoms might be. Treatments that reduce these hormones are being explored.

Antiandrogens might help prevent or treat COVID-19 by blocking the virus's entry into cells.

The drug enzalutamide may reduce the ability of the virus causing COVID-19 to enter lung cells.

The conclusion is that certain genetic factors and blood types may affect COVID-19 severity, but changes in ACE2 and TMPRSS2 genes are not clearly linked to it.

Spironolactone might help protect against severe lung problems in COVID-19 patients.

Existing drug dexamethasone may lower death risk in severe COVID-19 cases; more research needed for other drugs.

Testosterone may have a dual role in COVID-19, potentially worsening outcomes in men, and testosterone therapy could help some patients, but more research is needed.

COVID-19 may harm male fertility and damage the reproductive system.

Certain genetic differences may affect how likely someone is to get COVID-19 and how severe it might be.

Men and women get COVID-19 at similar rates, but men tend to get sicker and have a higher risk of dying, while women usually have stronger immune responses and vaccine reactions.

Androgens and a high-fat diet may increase the risk of severe COVID-19 in women with PCOS by upregulating certain proteins in the heart and kidneys.

Men with higher androgen levels may have severe COVID-19 symptoms, but those on antiandrogen therapy are less likely to contract the virus and have milder symptoms. Anti-androgen drugs could potentially treat COVID-19, but more research is needed.

Male pattern hair loss could hint at androgens affecting COVID-19 severity.

Androgenetic alopecia linked to COVID-19 severity; drugs reducing androgen receptor activation may help.