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Higher DHT levels are linked to fewer hypogonadism symptoms in men with normal testosterone levels.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

MG53 helps reduce skin damage caused by nitrogen mustard.

The document concludes that over 500 genes are linked to hair disorders and this knowledge is important for creating new treatments.

A defective protein in progeria causes cell death and atherosclerosis, but a treatment targeting cell stress may reduce these effects.

Hair greying is caused by oxidative stress damaging hair follicles and melanocytes.

Adenosine receptors could be promising targets for treating inflammatory skin diseases like psoriasis.

Melanocytes produce melanin; their defects cause vitiligo and hair graying, with treatments available for vitiligo.

Stem cell niches are adaptable and key for tissue maintenance and repair.

Gene therapy has potential as a future treatment for Hutchinson-Gilford progeria syndrome.

N-acetyl-L-cysteine (NAC) can prevent DNA damage and protect cells from harm.

TLR2 is important for hair growth and can be targeted to treat hair loss.

Vav2 changes how hair follicle stem cells' genes work as they age, which might improve regeneration but also raise cancer risk.

The European Society of Endocrinology advises individualized long-term management for PCOS, focusing on lifestyle changes, accurate diagnosis, and treatments for associated health risks and symptoms.

More dermal papilla cells in hair follicles lead to larger, healthier hair, while fewer cells cause hair thinning and loss.

Menopausal women have higher androgen levels, which can cause symptoms like facial hair growth and low sexual desire.

Alopecia areata involves immune response and gene changes affecting hair loss.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

Genetic mutations cause various hair diseases, and whole genome sequencing may reveal more about these conditions.

Patients with Systemic Sclerosis have much higher levels of GDF-15, which could help predict organ involvement and guide treatment.

The document concludes that alopecia has significant social and psychological effects, leading to a market for hair loss treatments.

The WNT signaling pathway is important in many diseases and targeting it could offer new treatments.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

The AUTS2 gene is linked to neurological disorders and may affect human brain development and cognition.

Androgen receptors are key for development and health, affecting conditions like prostate cancer and male pattern baldness.

Wnt10b makes hair follicles bigger, but DKK1 can reverse this effect.

The conclusion is that oral contraceptives and antiandrogens can treat hirsutism and acne in women with cutaneous hyperandrogenism, but more research is needed for effective treatments, especially for hair loss.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

Researchers found two genes that may explain why some Casertana pigs don't have hair.

Menopause often leads to lower sexual desire and discomfort during sex, but treatment should be personalized and only if it bothers the woman.