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Researchers found four genes that could help diagnose severe alopecia areata early.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

The document concludes that stem cell therapies lack solid proof of effectiveness, except for blood system treatments, and criticizes the ethical issues and commercial exploitation in the field.

New treatments for skin and hair repair show promise, but further improvements are needed.

The dermal regeneration template is effective in skin regeneration, reducing scarring, and has potential for future improvements.

Wnt3a activates certain genes in hair follicle cells, including a newly discovered one, EP2, which may affect hair growth.

Genes linked to fibrosis are more active in people with central centrifugal cicatricial alopecia.

Using micro skin tissue columns improves skin wound healing and reduces scarring.

Vitamin C derivative reduces hair loss-related protein in cells.

Adult esophageal cells can start to become like skin cells, with a key pathway influencing this change.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Regulatory γδ T cells help protect hair follicles from alopecia areata and promote hair regrowth.

Topical eye β-blockers may cause hair loss.