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Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Skin health and repair depend on the signals between skin stem cells and their surrounding cells.

Prominin-1 expressing cells in the dermal papilla help regulate hair follicle size and communication but don't aid in skin repair.

Higher p63 and CD34 levels found in specific scalp areas may affect hair loss progression.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

Nonpalmoplantar skin cells can be made to express keratin 9 by interacting with palmoplantar fibroblasts.

Fibroblast growth factors are crucial for hair follicle development and regeneration.

The dermal papilla is crucial for hair growth and health, and understanding it could lead to new hair loss treatments.

The document concludes that specific cells are essential for hair growth and more research is needed to understand how to maintain their hair-inducing properties.

A device was made in 2008 to measure hair loss severity. Other findings include: frizzy mutation in mice isn't related to Fgfr2, C/EBPx marks preadipocytes, Cyclosporin A speeds up hair growth in mice, blocking plasmin and metalloproteinases hinders healing, hyperbaric oxygen helps ischemic wound healing, amniotic membranes heal wounds better than polyurethane foam, rhVEGF165 from a fibrin matrix improves tissue flap viability and induces VEGF-R2 expression, and bFGF enhances wound healing and reduces scarring in rabbits.

Some wound-healing cells can turn into fat cells around new hair growth in mice.

Scientists made stem cells that can grow hair by adding three specific factors to them.

Transplanted baby mouse skin cells grew normal hair using a new, efficient method.

Small molecule DMF improves psoriasis and multiple sclerosis, adult skin cells can be made to grow new hair, certain skin cells initiate hair growth, IL-17C controls gut health and can cause skin inflammation, and skin cells produce IL-17 that can lead to psoriasis.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Hair follicle development and microbes help regulatory T cells gather in newborn skin.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

Different species and human skin models vary in their skin enzyme activities, with pig skin and some models closely matching human skin, useful for safety assessments and understanding the skin's protective roles.

Twist2 is essential for scarless skin healing and hair growth in mouse fetuses.

Twist2 is essential for proper skin healing and hair growth in developing mice.

Beige and leaden pigment genes act within melanocytes, affecting pigment patterns.

Some vitamin D analogs can thicken skin and reduce pore size like a common acne treatment, with one analog also affecting skin growth factors.

Researchers identified specific genes that are important for mouse skin cell development and healing.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

Adding a specific gene to skin cells can help treat skin disorders like psoriasis.

The research reveals how early embryonic mouse skin develops from simple to complex structures, identifying various cell types and their roles in this process.

The Sonic hedgehog pathway is crucial for new hair growth during mouse skin healing.

Mice can grow new hair follicles after skin wounds through a process not involving existing hair stem cells, but requiring more research to understand fully.