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Cannabinoid receptor-1 signaling is essential for the survival and growth of human hair follicle stem cells.

Progesterone and its byproducts control a specific receptor in the brain independently of progesterone receptors, affecting conditions related to the menstrual cycle.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Hair and skin can regenerate without bulge stem cells due to other compensating cells.

Blocking the mineralocorticoid receptor can help treat skin thinning caused by steroids.

Blocking DGAT1 reduces oil gland size in mice and dogs, but only mice experience hair loss.

Vitamin D Receptor is crucial for hair follicle shrinkage and cell death, affecting hair growth.

Erythropoietin overexpression disrupts hair growth and fat formation in mice.

mEphA1 receptor tyrosine kinase is important for skin and hair development and may play a role in certain diseases.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

CaBP1 and 2 are important for maintaining the activity of calcium channels necessary for hearing in inner ear cells.

A protein from fat-derived stem cells, DKK1, is linked to hair loss and blocking it may help treat alopecia areata.

Sdr16c5 and Sdr16c6 genes regulate a key point in lipid production that affects eye and skin gland function.

Mouse touch-sensitive nerve cells adjust their connections based on competition with other similar cells.

Dkk4 is necessary for the initial development and arrangement of hair follicles.

The research found that a specific skin cell type not only triggers hair growth but also controls hair color, and that aging can lead to hair loss and color changes.

Activating TLR3 helps improve skin and hair follicle regeneration after wounds.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

γδ T cells help with hair growth during wound healing in mice.

Follistatin is important for hair growth and could help treat hair loss.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

The timing of when the gene Bmal1 is active affects aging and survival, with its absence during development, not adulthood, leading to premature aging.

Mice with LSS deficiency showed hair loss and cataracts, similar to humans, and can help in understanding and treating this condition.

Mice without the IL-6 gene had more hair growth after injury due to higher activity of a related protein, Stat3.

Removing certain brain receptors in mice worsens seizure severity and response to treatment during hormone withdrawal.

Blocking YAP/TAZ could be a new way to treat skin cancer.

GRK2 is essential for healthy hair follicle function, and its absence can lead to hair loss and cysts.

Tet1/2/3 enzymes affect hair follicle cell development by influencing BMP signaling.

Removing PLCg1 from skin cells caused thicker oil glands and less hair in mice.